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Autophagy|January 19, 2021
Autophagy activation, lipotoxicity and lysosomal membrane permeabilization synergize to promote pimozide- and loperamide-induced glioma cell deathNina Meyer, Lisa Henkel, Benedikt Linder, et al.
Journal of Cellular and Molecular Medicine|February 21, 2009
Current evidence for a modulation of low back pain by human genetic variantsIrmgard Tegeder, Jörn Lötsch
International Journal of Molecular Sciences|September 10, 2021
Failure of Diphtheria Toxin Model to Induce Parkinson-Like Behavior in MiceLucie Valek, Irmgard Tegeder
Antioxidants (Basel, Switzerland)|April 3, 2021
Nucleoredoxin Knockdown in SH-SY5Y Cells Promotes Cell RenewalLucie Valek, Irmgard Tegeder
Acta Pharmacologica Sinica|December 9, 2025
Modest improvement of metabolic and behavioral deficits with long-term ambroxol treatment in a Pink1-/-SNCAA53T double mutant mouse model of Parkinson's diseaseLuisa Franck, Lucie Valek, Lisa Hahnefeld, et al.
Matrix Biology : Journal of the International Society for Matrix Biology|December 8, 2020
When lipid homeostasis runs havoc: Lipotoxicity links lysosomal dysfunction to autophagyIrmgard Tegeder, Donat Kögel
Journal of Visualized Experiments : Jove|March 14, 2017
Bioluminescence and Near-infrared Imaging of Optic Neuritis and Brain Inflammation in the EAE Model of Multiple Sclerosis in MiceKatja Schmitz, Irmgard Tegeder
Clinical Chemistry|March 17, 2007
Reliable screening for a pain-protective haplotype in the GTP cyclohydrolase 1 gene (GCH1) through the use of 3 or fewer single nucleotide polymorphismsJörn Lötsch, Inna Belfer, Anja Kirchhof, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|August 22, 2008
cGMP produced by NO-sensitive guanylyl cyclase essentially contributes to inflammatory and neuropathic pain by using targets different from cGMP-dependent protein kinase IAchim Schmidtko, Wei Gao, Peter König, et al.
Journal of Immunology (Baltimore, Md. : 1950)|August 5, 2011
The protein kinase IKKε is a potential target for the treatment of inflammatory hyperalgesiaChristine V Möser, Katharina Kynast, Katharina Baatz, et al.
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