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Isaac G Onyango

Showing results (1-10 of 23) with videos related to

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Neural Regeneration Research|February 17, 2018
Modulation of mitochondrial bioenergetics as a therapeutic strategy in Alzheimer's diseaseIsaac G Onyango
Neurochemical Research|October 18, 2007
Mitochondrial dysfunction and oxidative stress in Parkinson's diseaseIsaac G Onyango
Biomedicines|March 6, 2021
Energy, Entropy and Quantum Tunneling of Protons and Electrons in Brain Mitochondria: Relation to Mitochondrial Impairment in Aging-Related Human Brain Diseases and Therapeutic MeasuresJames P Bennett, Isaac G Onyango
Current Alzheimer Research|October 5, 2006
Oxidative stress, mitochondrial dysfunction, and stress signaling in Alzheimer's diseaseIsaac G Onyango, Shaharyar M Khan
Aging and Disease|April 27, 2016
Mitochondrial Dysfunction in Alzheimer's Disease and the Rationale for Bioenergetics Based TherapiesIsaac G Onyango, Jameel Dennis, Shaharyah M Khan
Frontiers in Bioscience (Landmark Edition)|November 5, 2016
Mitochondria in the pathophysiology of Alzheimer's and Parkinson's diseasesIsaac G Onyango, Shaharyar M Khan, James P Bennett
Neurobiology of Disease|September 3, 2005
Brain-derived growth factor and glial cell line-derived growth factor use distinct intracellular signaling pathways to protect PD cybrids from H2O2-induced neuronal deathIsaac G Onyango, Jeremy B Tuttle, James P Bennett
Molecular and Cellular Neurosciences|March 2, 2005
Activation of p38 and N-acetylcysteine-sensitive c-Jun NH2-terminal kinase signaling cascades is required for induction of apoptosis in Parkinson's disease cybridsIsaac G Onyango, Jeremy B Tuttle, James P Bennett
Neural Regeneration Research|January 12, 2021
Regulation of neuronal bioenergetics as a therapeutic strategy in neurodegenerative diseasesIsaac G Onyango, James P Bennett, Gorazd B Stokin
Neurobiology of Disease|April 20, 2005
Endogenous oxidative stress in sporadic Alzheimer's disease neuronal cybrids reduces viability by increasing apoptosis through pro-death signaling pathways and is mimicked by oxidant exposure of control cybridsIsaac G Onyango, James P Bennett, Jeremy B Tuttle
Pageof 3

Showing results (1-10 of 23) with videos related to

Sort By:
Pageof 3
Neural Regeneration Research|February 17, 2018
Modulation of mitochondrial bioenergetics as a therapeutic strategy in Alzheimer's diseaseIsaac G Onyango
Neurochemical Research|October 18, 2007
Mitochondrial dysfunction and oxidative stress in Parkinson's diseaseIsaac G Onyango
Biomedicines|March 6, 2021
Energy, Entropy and Quantum Tunneling of Protons and Electrons in Brain Mitochondria: Relation to Mitochondrial Impairment in Aging-Related Human Brain Diseases and Therapeutic MeasuresJames P Bennett, Isaac G Onyango
Current Alzheimer Research|October 5, 2006
Oxidative stress, mitochondrial dysfunction, and stress signaling in Alzheimer's diseaseIsaac G Onyango, Shaharyar M Khan
Aging and Disease|April 27, 2016
Mitochondrial Dysfunction in Alzheimer's Disease and the Rationale for Bioenergetics Based TherapiesIsaac G Onyango, Jameel Dennis, Shaharyah M Khan
Frontiers in Bioscience (Landmark Edition)|November 5, 2016
Mitochondria in the pathophysiology of Alzheimer's and Parkinson's diseasesIsaac G Onyango, Shaharyar M Khan, James P Bennett
Neurobiology of Disease|September 3, 2005
Brain-derived growth factor and glial cell line-derived growth factor use distinct intracellular signaling pathways to protect PD cybrids from H2O2-induced neuronal deathIsaac G Onyango, Jeremy B Tuttle, James P Bennett
Molecular and Cellular Neurosciences|March 2, 2005
Activation of p38 and N-acetylcysteine-sensitive c-Jun NH2-terminal kinase signaling cascades is required for induction of apoptosis in Parkinson's disease cybridsIsaac G Onyango, Jeremy B Tuttle, James P Bennett
Neural Regeneration Research|January 12, 2021
Regulation of neuronal bioenergetics as a therapeutic strategy in neurodegenerative diseasesIsaac G Onyango, James P Bennett, Gorazd B Stokin
Neurobiology of Disease|April 20, 2005
Endogenous oxidative stress in sporadic Alzheimer's disease neuronal cybrids reduces viability by increasing apoptosis through pro-death signaling pathways and is mimicked by oxidant exposure of control cybridsIsaac G Onyango, James P Bennett, Jeremy B Tuttle
Pageof 3