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Progress in Neurobiology|February 27, 2013
Clinical features, neurogenetics and neuropathology of the polyglutamine spinocerebellar ataxias type 1, 2, 3, 6 and 7Udo Rüb, Ludger Schöls, Henry Paulson, et al.
Redox Biology|November 10, 2017
Pharmacological targeting of GSK-3 and NRF2 provides neuroprotection in a preclinical model of tauopathyAntonio Cuadrado, Sebastian Kügler, Isabel Lastres-Becker
Current Opinion in Neurology|April 11, 2009
Role of microglial redox balance in modulation of neuroinflammationNadia G Innamorato, Isabel Lastres-Becker, Antonio Cuadrado
Antioxidants (Basel, Switzerland)|August 26, 2023
Aging, NRF2, and TAU: A Perfect Match for Neurodegeneration?Mirjam Brackhan, Marina Arribas-Blazquez, Isabel Lastres-Becker
Journal of Neuroimmunology|January 28, 2006
Endotoxin preconditioning protects neurones from in vitro ischemia: role of endogenous IL-1beta and TNF-alphaIsabel Lastres-Becker, Tam Cartmell, Francisco Molina-Holgado
International Journal of Molecular Sciences|February 13, 2026
Bioinformatic Analyses of the Ataxin-2 Family Since Algae Emphasize Its Small Isoforms, Large Chimerisms, and the Importance of Human Exon 1B as Target of Therapies to Prevent NeurodegenerationGeorg W J Auburger, Jana Key, Suzana Gispert, et al.
Neurobiology of Disease|May 21, 2026
Analysisoftheinvolvementof RNA-bindingproteinsintau-dependentneurodegenerationIgnacio Silva-Llanes, Pablo Baceiredo-Macho, Isabel Lastres-Becker
The Journal of Biological Chemistry|April 25, 2014
Transcription factors NRF2 and NF-κB are coordinated effectors of the Rho family, GTP-binding protein RAC1 during inflammationAntonio Cuadrado, Zaira Martín-Moldes, Jianping Ye, et al.
Frontiers in Neuroscience|February 11, 2025
RIPK1 expression and inhibition in tauopathies: implications for neuroinflammation and neuroprotectionIgnacio Silva-Llanes, Enrique Madruga, Ana Martínez, et al.
Molecular and Cellular Neurosciences|August 19, 2008
Pituitary adenylate cyclase-activating polypeptide stimulates glial fibrillary acidic protein gene expression in cortical precursor cells by activating Ras and Rap1Isabel Lastres-Becker, Antonio Fernández-Pérez, Beatriz Cebolla, et al.
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