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Frontiers in Aging Neuroscience
|
July 30, 2025
Dysregulated calcium signaling in the aged primate association cortices: vulnerability to Alzheimer's disease neuropathology
Amy F T Arnsten, Isabella Perone, Min Wang, et al.
Alzheimer'S & Dementia : the Journal of the Alzheimer'S Association
|
December 24, 2025
Basic Science and Pathogenesis
Dibyadeep Datta, Dinara Bolat, Isabella Perone, et al.
Neuromolecular Medicine
|
June 24, 2022
Mitochondrial SIRT3 Deficiency Results in Neuronal Network Hyperexcitability, Accelerates Age-Related Aβ Pathology, and Renders Neurons Vulnerable to Aβ Toxicity
Isabella Perone, Nathaniel Ghena, Jing Wang, et al.
Cerebral Cortex (New York, N.Y. : 1991)
|
October 24, 2023
Localization of PDE4D, HCN1 channels, and mGluR3 in rhesus macaque entorhinal cortex may confer vulnerability in Alzheimer's disease
Dibyadeep Datta, Isabella Perone, Yury M Morozov, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience
|
December 11, 2019
SIRT3 Haploinsufficiency Aggravates Loss of GABAergic Interneurons and Neuronal Network Hyperexcitability in an Alzheimer's Disease Model
Aiwu Cheng, Jing Wang, Nathaniel Ghena, et al.
Biorxiv : the Preprint Server for Biology
|
November 21, 2023
Nanoscale imaging of pT217-tau in aged rhesus macaque entorhinal and dorsolateral prefrontal cortex: Evidence of interneuronal trafficking and early-stage neurodegeneration
Dibyadeep Datta, Isabella Perone, Denethi Wijegunawardana, et al.
Alzheimer'S & Dementia : the Journal of the Alzheimer'S Association
|
March 6, 2024
Nanoscale imaging of pT217-tau in aged rhesus macaque entorhinal and dorsolateral prefrontal cortex: Evidence of interneuronal trafficking and early-stage neurodegeneration
Dibyadeep Datta, Isabella Perone, Denethi Wijegunawardana, et al.
JAMA Psychiatry
|
May 22, 2024
Key Roles of CACNA1C/Cav1.2 and CALB1/Calbindin in Prefrontal Neurons Altered in Cognitive Disorders
Dibyadeep Datta, Shengtao Yang, Mary Kate P Joyce, et al.
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of 1
Search research articles
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Showing results (1-10 of 8) with videos related to
Sort By:
Page
of 1
Frontiers in Aging Neuroscience
|
July 30, 2025
Dysregulated calcium signaling in the aged primate association cortices: vulnerability to Alzheimer's disease neuropathology
Amy F T Arnsten, Isabella Perone, Min Wang, et al.
Alzheimer'S & Dementia : the Journal of the Alzheimer'S Association
|
December 24, 2025
Basic Science and Pathogenesis
Dibyadeep Datta, Dinara Bolat, Isabella Perone, et al.
Neuromolecular Medicine
|
June 24, 2022
Mitochondrial SIRT3 Deficiency Results in Neuronal Network Hyperexcitability, Accelerates Age-Related Aβ Pathology, and Renders Neurons Vulnerable to Aβ Toxicity
Isabella Perone, Nathaniel Ghena, Jing Wang, et al.
Cerebral Cortex (New York, N.Y. : 1991)
|
October 24, 2023
Localization of PDE4D, HCN1 channels, and mGluR3 in rhesus macaque entorhinal cortex may confer vulnerability in Alzheimer's disease
Dibyadeep Datta, Isabella Perone, Yury M Morozov, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience
|
December 11, 2019
SIRT3 Haploinsufficiency Aggravates Loss of GABAergic Interneurons and Neuronal Network Hyperexcitability in an Alzheimer's Disease Model
Aiwu Cheng, Jing Wang, Nathaniel Ghena, et al.
Biorxiv : the Preprint Server for Biology
|
November 21, 2023
Nanoscale imaging of pT217-tau in aged rhesus macaque entorhinal and dorsolateral prefrontal cortex: Evidence of interneuronal trafficking and early-stage neurodegeneration
Dibyadeep Datta, Isabella Perone, Denethi Wijegunawardana, et al.
Alzheimer'S & Dementia : the Journal of the Alzheimer'S Association
|
March 6, 2024
Nanoscale imaging of pT217-tau in aged rhesus macaque entorhinal and dorsolateral prefrontal cortex: Evidence of interneuronal trafficking and early-stage neurodegeneration
Dibyadeep Datta, Isabella Perone, Denethi Wijegunawardana, et al.
JAMA Psychiatry
|
May 22, 2024
Key Roles of CACNA1C/Cav1.2 and CALB1/Calbindin in Prefrontal Neurons Altered in Cognitive Disorders
Dibyadeep Datta, Shengtao Yang, Mary Kate P Joyce, et al.
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