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Blood|November 8, 2019
Description of a knock-in mouse model of JAK2V617F MPN emerging from a minority of mutated hematopoietic stem cellsOlivier Mansier, Badr Kilani, Amélie V Guitart, et al.
Hemasphere|April 6, 2026
Unraveling the role of <i>TP53</i> mutations in myeloproliferative neoplasms: Molecular mechanisms of leukemic transformationSuzana da Silva-Benedito, Paula Sabbo Bernardo, Jean-Edouard Martin, et al.
Haematologica|December 23, 2017
New pathogenic mechanisms induced by germline erythropoietin receptor mutations in primary erythrocytosisFlorence Pasquier, Caroline Marty, Thomas Balligand, et al.
F1000Research|May 3, 2016
Recent advances in understanding myelofibrosis and essential thrombocythemiaWilliam Vainchenker, Stefan N Constantinescu, Isabelle Plo
The Journal of Pharmacology and Experimental Therapeutics|June 30, 2023
Optimizing IFN Alpha Therapy against Myeloproliferative NeoplasmsGurvan Hermange, Paul-Henry Cournède, Isabelle Plo
Molecular & Cellular Oncology|June 17, 2016
ATG2B and GSKIP: 2 new genes predisposing to myeloid malignanciesIsabelle Plo, Christine Bellanné-Chantelot, William Vainchenker
Blood|November 27, 2015
Calreticulin mutants in mice induce an MPL-dependent thrombocytosis with frequent progression to myelofibrosisCaroline Marty, Christian Pecquet, Harini Nivarthi, et al.
Oncotarget|March 10, 2016
P53 activation inhibits all types of hematopoietic progenitors and all stages of megakaryopoiesisEmna Mahfoudhi, Larissa Lordier, Caroline Marty, et al.
Oncogene|March 9, 2019
Rare type 1-like and type 2-like calreticulin mutants induce similar myeloproliferative neoplasms as prevalent type 1 and 2 mutants in miceKatte Rao Toppaldoddi, Maira da Costa Cacemiro, Olivier Bluteau, et al.
Mathematical Medicine and Biology : a Journal of the IMA|June 14, 2024
Mathematical modelling, selection and hierarchical inference to determine the minimal dose in IFNα therapy against myeloproliferative neoplasmsGurvan Hermange, William Vainchenker, Isabelle Plo, et al.
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