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Acta Neuropathologica Communications|June 23, 2021
Peripheral administration of SOD1 aggregates does not transmit pathogenic aggregation to the CNS of SOD1 transgenic miceIsil Keskin, Elaheh Ekhtiari Bidhendi, Matthew Marklund, et al.Plos One|February 27, 2016
Effects of Cellular Pathway Disturbances on Misfolded Superoxide Dismutase-1 in Fibroblasts Derived from ALS PatientsIsil Keskin, Elin Forsgren, Dale J Lange, et al.Amyotrophic Lateral Sclerosis & Frontotemporal Degeneration|March 23, 2017
Comprehensive analysis to explain reduced or increased SOD1 enzymatic activity in ALS patients and their relativesIsil Keskin, Anna Birve, Mariusz Berdynski, et al.Acta Neuropathologica|March 14, 2019
The molecular pathogenesis of superoxide dismutase 1-linked ALS is promoted by low oxygen tensionIsil Keskin, Elin Forsgren, Manuela Lehmann, et al.Human Molecular Genetics|May 19, 2012
Mutant superoxide dismutase-1 indistinguishable from wild-type causes ALSMatthis Synofzik, Dario Ronchi, Isil Keskin, et al.Alzheimer'S & Dementia : the Journal of the Alzheimer'S Association|April 28, 2025
A comparison of p-tau assays for the specificity to detect tau changes in Alzheimer's diseaseShorena Janelidze, Nicholas J Ashton, Anna Orduña Dolado, et al.Brain Communications|February 16, 2023
The motor system is exceptionally vulnerable to absence of the ubiquitously expressed superoxide dismutase-1Julien H Park, Ulrika Nordström, Konstantinos Tsiakas, et al.Pageof 1