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Frontiers in Immunology|August 17, 2019
The CXCL12/CXCR4 Signaling Axis Retains Neutrophils at Inflammatory Sites in ZebrafishHannah M Isles, Kimberly D Herman, Anne L Robertson, et al.
Molecular Brain|December 10, 2016
Increased alternate splicing of Htr2c in a mouse model for Prader-Willi syndrome leads disruption of 5HT2C receptor mediated appetiteAlastair S Garfield, Jennifer R Davies, Luke K Burke, et al.
Genes, Brain, and Behavior|September 1, 2017
Dopaminergic and behavioural changes in a loss-of-imprinting model of Cdkn1cG I McNamara, B A Davis, M Browne, et al.
Molecular and Cellular Biology|March 31, 2005
Imprinted Nesp55 influences behavioral reactivity to novel environmentsAntonius Plagge, Anthony R Isles, Emma Gordon, et al.
Nature Genetics|May 24, 2005
Xlr3b is a new imprinted candidate for X-linked parent-of-origin effects on cognitive function in miceWilliam Davies, Anthony Isles, Rachel Smith, et al.
The New England Journal of Medicine|November 16, 1995
Prevention of coronary heart disease with pravastatin in men with hypercholesterolemia. West of Scotland Coronary Prevention Study GroupJ Shepherd, S M Cobbe, I Ford, et al.
Human Molecular Genetics|November 30, 2017
Loss of offspring Peg3 reduces neonatal ultrasonic vocalizations and increases maternal anxiety in wild-type mothersG I McNamara, H D J Creeth, D J Harrison, et al.
British Journal of Clinical Pharmacology|March 1, 1984
Effect of the 5-hydroxytryptamine type 2 receptor antagonist, ketanserin, on blood pressure, the renin-angiotensin system and sympatho-adrenal function in patients with essential hypertensionJ R Zabludowski, C Zoccali, C G Isles, et al.
Cell Genomics|February 15, 2024
Better translation via collaboration: The MRC National Mouse Genetics NetworkOwen Sansom, Sara Wells, David Kent, et al.
British Journal of Clinical Pharmacology|September 1, 1983
The effect of a 5-HT antagonist, ketanserin, on blood pressure, the renin-angiotensin system and sympathoadrenal function in normal manC Zoccali, J R Zabludowski, C G Isles, et al.
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