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Journal of Molecular and Cellular Cardiology|August 1, 2024
Macrophages in the infarcted heart acquire a fibrogenic phenotype, expressing matricellular proteins, but do not undergo fibroblast conversionRuoshui Li, Anis Hanna, Shuaibo Huang, et al.
European Heart Journal|August 24, 2005
Cells of primarily extra-valvular origin in degenerative aortic valves and bioprosthesesDirk Skowasch, Stephanie Schrempf, Nicolas Wernert, et al.
Circulation Research|June 20, 2024
Fibroblast Smad7 Induction Protects the Remodeling Pressure-Overloaded HeartClaudio Humeres, Arti V Shinde, Izabela Tuleta, et al.
Bioscience Reports|August 21, 2019
Diaphragm function does not independently predict exercise intolerance in patients with precapillary pulmonary hypertension after adjustment for right ventricular functionJens Spiesshoefer, Simon Herkenrath, Michael Mohr, et al.
Sleep & Breathing = Schlaf & Atmung|July 24, 2020
Effects of central apneas on sympathovagal balance and hemodynamics at night: impact of underlying systolic heart failureJens Spiesshoefer, Nora Hegerfeld, Malte Frank Gerdes, et al.
Plos One|September 14, 2016
Pseudoxanthoma Elasticum - Also a Lung Disease? The Respiratory Affection of Patients with Pseudoxanthoma ElasticumSimon Pingel, Sebastian Gorgonius Passon, Kristin Solveig Pausewang, et al.
VASA. Zeitschrift Fur Gefasskrankheiten|December 9, 2016
Increased vascular occlusion in patients with pseudoxanthoma elasticumSimon Pingel, Kristin Solveig Pausewang, Sebastian Gorgonius Passon, et al.
Respiration; International Review of Thoracic Diseases|November 10, 2020
Heart Failure Results in Inspiratory Muscle Dysfunction Irrespective of Left Ventricular Ejection FractionJens Spiesshoefer, Carolin Henke, Hans Joachim Kabitz, et al.
Nature Communications|November 21, 2023
Protective effects of macrophage-specific integrin α5 in myocardial infarction are associated with accentuated angiogenesisRuoshui Li, Bijun Chen, Akihiko Kubota, et al.
Science Translational Medicine|November 3, 2021
Truncated titin proteins and titin haploinsufficiency are targets for functional recovery in human cardiomyopathy due to <i>TTN</i> mutationsAndrey Fomin, Anna Gärtner, Lukas Cyganek, et al.
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