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Neurological Research|April 19, 2007
Endothelial-neutrophil interactions during ischemia and reperfusion injury: basic mechanisms of hyperbaric oxygenJ A Buras, W R ReenstraJournal of Immunology (Baltimore, Md. : 1950)|August 15, 1992
IL-4 reciprocally regulates IL-1 and IL-1 receptor antagonist expression in human monocytesM J Fenton, J A Buras, R P DonnellyMolecular Immunology|June 1, 1995
NF beta A, a factor required for maximal interleukin-1 beta gene expression is identical to the ets family member PU.1. Evidence for structural alteration following LPS activationJ A Buras, W R Reenstra, M J FentonJournal of Immunology (Baltimore, Md. : 1950)|May 1, 1994
The NF-beta A-binding element, not an overlapping NF-IL-6-binding element, is required for maximal IL-1 beta gene expressionJ A Buras, B G Monks, M J FentonAmerican Journal of Physiology. Cell Physiology|February 9, 2000
Hyperbaric oxygen downregulates ICAM-1 expression induced by hypoxia and hypoglycemia: the role of NOSJ A Buras, G L Stahl, K K Svoboda, et al.Molecular Immunology|February 1, 1994
An upstream protein interacts with a distinct protein that binds to the cap site of the human interleukin 1 beta geneB G Monks, B A Martell, J A Buras, et al.The American Journal of Pathology|September 11, 2001
Endothelial oxidative stress activates the lectin complement pathway: role of cytokeratin 1C D Collard, M C Montalto, W R Reenstra, et al.Journal of Immunology (Baltimore, Md. : 1950)|March 10, 2001
A keratin peptide inhibits mannose-binding lectinM C Montalto, C D Collard, J A Buras, et al.The American Journal of Pathology|May 4, 2000
Complement activation after oxidative stress: role of the lectin complement pathwayC D Collard, A Väkevä, M A Morrissey, et al.Pageof 1