J A Ember|T E Hugli - JoVE Visualize

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Biopolymers|January 1, 1996

Stabilization of an isolated helical capping box in solution by hydrophobic interactions: evidence from the NMR study of bioactive peptides from the C-terminus of human C5a anaphylatoxinF Ni, K A Carpenter, D R Ripoll, et al.

The Journal of Laboratory and Clinical Medicine|February 1, 1984

Plasma lactoferrin reflects granulocyte activation via complement in burn patientsB Wolach, T D Coates, T E Hugli, et al.

Journal of Immunology (Baltimore, Md. : 1950)|July 1, 1985

Salivary gland lymphocytes in primary Sjogren's syndrome lack lymphocyte subsets defined by Leu-7 and Leu-11 antigensR I Fox, T E Hugli, L L Lanier, et al.

The Journal of Biological Chemistry|March 10, 1985

Evidence for a role of the amino-terminal region in the biological activity of the classical anaphylatoxin, porcine C5a des-Arg-74C Gerard, H J Showell, P D Hoeprich, et al.

The Journal of Clinical Investigation|September 1, 1979

Anaphylatoxin-induced histamine release with human leukocytes: studies of C3a leukocyte binding and histamine releaseM M Glovsky, T E Hugli, T Ishizaka, et al.

Immunopharmacology|May 1, 1994

Uptake of C5a by polymorphonuclear leukocytes (PMNs) after focal cerebral ischemia. I. Effect of tirilazad mesylate intervention on C5a uptake by PMNsG Hetland, G J del Zoppo, E Mori, et al.

Archives of Surgery (Chicago, Ill. : 1960)|December 1, 1988

Effects of cardiopulmonary bypass on pulmonary leukostasis and complement activationR J Howard, C Crain, D A Franzini, et al.

Molecular Immunology|June 1, 1983

The active site of human C4a anaphylatoxinT E Hugli, M S Kawahara, C G Unson, et al.

Journal of Immunology (Baltimore, Md. : 1950)|November 1, 1984

C3d-K, a kallikrein cleavage fragment of iC3b is a potent inhibitor of cellular proliferationM L Thoman, J L Meuth, E L Morgan, et al.

The Journal of Biological Chemistry|March 10, 1985

A synthetic nonapeptide corresponding to the NH2-terminal sequence of C3d-K causes leukocytosis in rabbitsP D Hoeprich, C A Dahinden, P J Lachmann, et al.

Pageof 18

Showing results (131-140 of 178) with videos related to

Sort By:

Pageof 18

Biopolymers|January 1, 1996

Stabilization of an isolated helical capping box in solution by hydrophobic interactions: evidence from the NMR study of bioactive peptides from the C-terminus of human C5a anaphylatoxinF Ni, K A Carpenter, D R Ripoll, et al.

The Journal of Laboratory and Clinical Medicine|February 1, 1984

Plasma lactoferrin reflects granulocyte activation via complement in burn patientsB Wolach, T D Coates, T E Hugli, et al.

Journal of Immunology (Baltimore, Md. : 1950)|July 1, 1985

Salivary gland lymphocytes in primary Sjogren's syndrome lack lymphocyte subsets defined by Leu-7 and Leu-11 antigensR I Fox, T E Hugli, L L Lanier, et al.

The Journal of Biological Chemistry|March 10, 1985

Evidence for a role of the amino-terminal region in the biological activity of the classical anaphylatoxin, porcine C5a des-Arg-74C Gerard, H J Showell, P D Hoeprich, et al.

The Journal of Clinical Investigation|September 1, 1979

Anaphylatoxin-induced histamine release with human leukocytes: studies of C3a leukocyte binding and histamine releaseM M Glovsky, T E Hugli, T Ishizaka, et al.

Immunopharmacology|May 1, 1994

Uptake of C5a by polymorphonuclear leukocytes (PMNs) after focal cerebral ischemia. I. Effect of tirilazad mesylate intervention on C5a uptake by PMNsG Hetland, G J del Zoppo, E Mori, et al.

Archives of Surgery (Chicago, Ill. : 1960)|December 1, 1988

Effects of cardiopulmonary bypass on pulmonary leukostasis and complement activationR J Howard, C Crain, D A Franzini, et al.

Molecular Immunology|June 1, 1983

The active site of human C4a anaphylatoxinT E Hugli, M S Kawahara, C G Unson, et al.

Journal of Immunology (Baltimore, Md. : 1950)|November 1, 1984

C3d-K, a kallikrein cleavage fragment of iC3b is a potent inhibitor of cellular proliferationM L Thoman, J L Meuth, E L Morgan, et al.

The Journal of Biological Chemistry|March 10, 1985

A synthetic nonapeptide corresponding to the NH2-terminal sequence of C3d-K causes leukocytosis in rabbitsP D Hoeprich, C A Dahinden, P J Lachmann, et al.

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