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J A Watts

Showing results (11-20 of 52) with videos related to

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Journal of Toxicology. Clinical Toxicology|March 17, 1999
Iron-mediated cardiotoxicity develops independently of extracellular hydroxyl radicals in isolated rat heartsJ A Watts, M D Ford, E Leonova
The American Journal of Physiology|November 1, 1981
Adenine nucleotide transport during cardiac ischemiaK F LaNoue, J A Watts, C D Koch
The American Journal of Physiology|June 1, 1980
Effects of Ca2+ antagonism on energy metabolism: Ca2+ and heart function after ischemiaJ A Watts, C D Koch, K F LaNoue
Journal of Applied Physiology (Bethesda, Md. : 1985)|April 12, 2001
Biventricular cardiac dysfunction after acute massive pulmonary embolism in the ratD M Sullivan, J A Watts, J A Kline
Journal of Molecular and Cellular Cardiology|August 1, 1985
Protection by verapamil of globally ischemic rat hearts: energy preservation, a partial explanationJ A Watts, L J Maiorano, P C Maiorano
Shock (Augusta, Ga.)|August 18, 2000
Depletion of lactate by dichloroacetate reduces cardiac efficiency after hemorrhagic shockR W Barbee, J A Kline, J A Watts
Shock (Augusta, Ga.)|December 10, 1999
A comparison of resuscitation with packed red blood cells and whole blood following hemorrhagic shock in caninesR W Barbee, J A Kline, J A Watts
Lancet (London, England)|September 26, 1981
Streptococcus pneumoniae resistant to penicillin and chloramphenicol in the U.KE W Williams, J A Watts, M R Potten
Journal of Molecular and Cellular Cardiology|March 1, 1986
Comparison of the protective effects of verapamil, diltiazem, nifedipine, and buffer containing low calcium upon global myocardial ischemic injuryJ A Watts, L J Maiorano, P C Maiorano
Journal of Molecular and Cellular Cardiology|August 1, 1987
A low concentration of nisoldipine reduces ischemic heart injury: enhanced reflow and recovery of contractile function without energy preservation during ischemiaJ A Watts, J P Whipple, A A Hatley
Pageof 6

Showing results (11-20 of 52) with videos related to

Sort By:
Pageof 6
Journal of Toxicology. Clinical Toxicology|March 17, 1999
Iron-mediated cardiotoxicity develops independently of extracellular hydroxyl radicals in isolated rat heartsJ A Watts, M D Ford, E Leonova
The American Journal of Physiology|November 1, 1981
Adenine nucleotide transport during cardiac ischemiaK F LaNoue, J A Watts, C D Koch
The American Journal of Physiology|June 1, 1980
Effects of Ca2+ antagonism on energy metabolism: Ca2+ and heart function after ischemiaJ A Watts, C D Koch, K F LaNoue
Journal of Applied Physiology (Bethesda, Md. : 1985)|April 12, 2001
Biventricular cardiac dysfunction after acute massive pulmonary embolism in the ratD M Sullivan, J A Watts, J A Kline
Journal of Molecular and Cellular Cardiology|August 1, 1985
Protection by verapamil of globally ischemic rat hearts: energy preservation, a partial explanationJ A Watts, L J Maiorano, P C Maiorano
Shock (Augusta, Ga.)|August 18, 2000
Depletion of lactate by dichloroacetate reduces cardiac efficiency after hemorrhagic shockR W Barbee, J A Kline, J A Watts
Shock (Augusta, Ga.)|December 10, 1999
A comparison of resuscitation with packed red blood cells and whole blood following hemorrhagic shock in caninesR W Barbee, J A Kline, J A Watts
Lancet (London, England)|September 26, 1981
Streptococcus pneumoniae resistant to penicillin and chloramphenicol in the U.KE W Williams, J A Watts, M R Potten
Journal of Molecular and Cellular Cardiology|March 1, 1986
Comparison of the protective effects of verapamil, diltiazem, nifedipine, and buffer containing low calcium upon global myocardial ischemic injuryJ A Watts, L J Maiorano, P C Maiorano
Journal of Molecular and Cellular Cardiology|August 1, 1987
A low concentration of nisoldipine reduces ischemic heart injury: enhanced reflow and recovery of contractile function without energy preservation during ischemiaJ A Watts, J P Whipple, A A Hatley
Pageof 6