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Peptides|September 1, 1987
Isolation of multiple biologically and chemically diverse species of epidermal growth factorM W Riemen, R J Wegrzyn, A E Baker, et al.
Bioorganic & Medicinal Chemistry|January 1, 1997
Cloning and mutagenesis of the p110 alpha subunit of human phosphoinositide 3'-hydroxykinaseS M Stirdivant, J Ahern, R R Conroy, et al.
Molecular and Cellular Biology|September 1, 1989
Substitution of lysine for arginine at position 42 of human transforming growth factor-alpha eliminates biological activity without changing internal disulfide bondsD Defeo-Jones, J Y Tai, G A Vuocolo, et al.
Oncogene|December 1, 1991
Dominant inhibitory Ras mutants demonstrate the requirement for Ras activity in the action of tyrosine kinase oncogenesD W Stacey, M Roudebush, R Day, et al.
Molecular and Cellular Biology|May 1, 1991
Ras GTPase-activating protein physically associates with mitogenically active phospholipidsM H Tsai, M Roudebush, S Dobrowolski, et al.
Oncogene|May 1, 1991
Mammalian cell lines engineered to identify inhibitors of specific signal transduction pathwaysR E Jones, D Defeo-Jones, E M McAvoy, et al.
Molecular and Cellular Biology|July 1, 1989
Epidermal growth factor receptor binding is affected by structural determinants in the toxin domain of transforming growth factor-alpha-Pseudomonas exotoxin fusion proteinsG M Edwards, D DeFeo-Jones, J Y Tai, et al.
Biochemistry|April 21, 1992
Steady-state kinetic mechanism of Ras farnesyl:protein transferaseD L Pompliano, E Rands, M D Schaber, et al.
Molecular and Cellular Biology|January 7, 1998
A farnesyltransferase inhibitor induces tumor regression in transgenic mice harboring multiple oncogenic mutations by mediating alterations in both cell cycle control and apoptosisR E Barrington, M A Subler, E Rands, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 21, 1998
Farnesyl transferase inhibitors cause enhanced mitotic sensitivity to taxol and epothilonesM M Moasser, L Sepp-Lorenzino, N E Kohl, et al.
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