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J B Schulz

Showing results (101-110 of 124) with videos related to

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BMC Geriatrics|June 15, 2018
Comprehensive pharmaceutical care to prevent drug-related readmissions of dependent-living elderly patients: a randomized controlled trialR Lenssen, K Schmitz, C Griesel, et al.
Brain : a Journal of Neurology|October 23, 2003
MRI-based volumetric differentiation of sporadic cerebellar ataxiaK Burk, C Globas, T Wahl, et al.
Molecular and Cellular Neurosciences|April 21, 2001
Cascade of caspase activation in potassium-deprived cerebellar granule neurons: targets for treatment with peptide and protein inhibitors of apoptosisE Gerhardt, S Kügler, M Leist, et al.
Brain Research|March 25, 1996
NGF, BDNF and NT-5, but not NT-3 protect against MPP+ toxicity and oxidative stress in neonatal animalsP B Kirschner, B G Jenkins, J B Schulz, et al.
Experimental Neurology|October 1, 1994
Systemic or local administration of azide produces striatal lesions by an energy impairment-induced excitotoxic mechanismE Brouillet, B T Hyman, B G Jenkins, et al.
Brain Research|April 27, 1999
Glutathione depletion and neuronal cell death: the role of reactive oxygen intermediates and mitochondrial functionU Wüllner, J Seyfried, P Groscurth, et al.
Experimental Cell Research|July 2, 1999
MPP+ inhibits proliferation of PC12 cells by a p21(WAF1/Cip1)-dependent pathway and induces cell death in cells lacking p21(WAF1/Cip1)F Soldner, M Weller, S Haid, et al.
Journal of Neurochemistry|August 1, 1995
3-Nitropropionic acid neurotoxicity is attenuated in copper/zinc superoxide dismutase transgenic miceM F Beal, R J Ferrante, R Henshaw, et al.
Brain : a Journal of Neurology|October 8, 1998
Autosomal dominant cerebellar ataxia type I. MRI-based volumetry of posterior fossa structures and basal ganglia in spinocerebellar ataxia types 1, 2 and 3T Klockgether, M Skalej, D Wedekind, et al.
Annals of Neurology|October 23, 1997
Increased 3-nitrotyrosine and oxidative damage in mice with a human copper/zinc superoxide dismutase mutationR J Ferrante, L A Shinobu, J B Schulz, et al.
Pageof 13

Showing results (101-110 of 124) with videos related to

Sort By:
Pageof 13
BMC Geriatrics|June 15, 2018
Comprehensive pharmaceutical care to prevent drug-related readmissions of dependent-living elderly patients: a randomized controlled trialR Lenssen, K Schmitz, C Griesel, et al.
Brain : a Journal of Neurology|October 23, 2003
MRI-based volumetric differentiation of sporadic cerebellar ataxiaK Burk, C Globas, T Wahl, et al.
Molecular and Cellular Neurosciences|April 21, 2001
Cascade of caspase activation in potassium-deprived cerebellar granule neurons: targets for treatment with peptide and protein inhibitors of apoptosisE Gerhardt, S Kügler, M Leist, et al.
Brain Research|March 25, 1996
NGF, BDNF and NT-5, but not NT-3 protect against MPP+ toxicity and oxidative stress in neonatal animalsP B Kirschner, B G Jenkins, J B Schulz, et al.
Experimental Neurology|October 1, 1994
Systemic or local administration of azide produces striatal lesions by an energy impairment-induced excitotoxic mechanismE Brouillet, B T Hyman, B G Jenkins, et al.
Brain Research|April 27, 1999
Glutathione depletion and neuronal cell death: the role of reactive oxygen intermediates and mitochondrial functionU Wüllner, J Seyfried, P Groscurth, et al.
Experimental Cell Research|July 2, 1999
MPP+ inhibits proliferation of PC12 cells by a p21(WAF1/Cip1)-dependent pathway and induces cell death in cells lacking p21(WAF1/Cip1)F Soldner, M Weller, S Haid, et al.
Journal of Neurochemistry|August 1, 1995
3-Nitropropionic acid neurotoxicity is attenuated in copper/zinc superoxide dismutase transgenic miceM F Beal, R J Ferrante, R Henshaw, et al.
Brain : a Journal of Neurology|October 8, 1998
Autosomal dominant cerebellar ataxia type I. MRI-based volumetry of posterior fossa structures and basal ganglia in spinocerebellar ataxia types 1, 2 and 3T Klockgether, M Skalej, D Wedekind, et al.
Annals of Neurology|October 23, 1997
Increased 3-nitrotyrosine and oxidative damage in mice with a human copper/zinc superoxide dismutase mutationR J Ferrante, L A Shinobu, J B Schulz, et al.
Pageof 13