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Molecular Psychiatry|January 23, 2002
Early experience and serotonin transporter gene variation interact to influence primate CNS functionA J Bennett, K P Lesch, A Heils, et al.
Journal of Medicinal Chemistry|May 27, 1994
CCK-A-selective tetrapeptides containing lys(N epsilon)-amide residues: favorable in vivo and in vitro effects of N-methylation at the aspartyl residueM J Bennett, A L Nikkel, B R Bianchi, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|December 9, 2024
Mitochondrial A3 Adenosine Receptor as a Mechanism for the Protective Effects of A3AR Agonists on Chemotherapy-Induced Neuropathic PainTimothy M Doyle, Kali Janes, Wen Hua Xiao, et al.
The Journal of Clinical Endocrinology and Metabolism|May 31, 2007
Detailed analysis of variation at and around mitochondrial position 16189 in a large Finnish cohort reveals no significant associations with early growth or metabolic phenotypes at age 31 yearsSreena Das, Amanda J Bennett, Ulla Sovio, et al.
Journal of Neurophysiology|January 10, 2019
Locomotor-related V3 interneurons initiate and coordinate muscles spasms after spinal cord injuryShihao Lin, Yaqing Li, Ana M Lucas-Osma, et al.
Proceedings. Mathematical, Physical, and Engineering Sciences|November 10, 2017
On three-dimensional misorientation spacesRobert Krakow, Robbie J Bennett, Duncan N Johnstone, et al.
Scientific Reports|July 31, 2015
Decrease of mRNA Editing after Spinal Cord Injury is Caused by Down-regulation of ADAR2 that is Triggered by Inflammatory ResponseAntonio Fabio Di Narzo, Alexey Kozlenkov, Yongchao Ge, et al.
Nature Cell Biology|December 24, 2013
A secreted peptide acts on BIN2-mediated phosphorylation of ARFs to potentiate auxin response during lateral root developmentHyunwoo Cho, Hojin Ryu, Sangchul Rho, et al.
Brain : a Journal of Neurology|June 4, 2018
Eliciting inflammation enables successful rehabilitative training in chronic spinal cord injuryAbel Torres-Espín, Juan Forero, Keith K Fenrich, et al.
Cell Reports|September 8, 2021
Persistent mRNA localization defects and cell death in ALS neurons caused by transient cellular stressSebastian Markmiller, Shashank Sathe, Kari L Server, et al.
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