Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

J C Brüning

Showing results (1-10 of 21) with videos related to

Pageof 3
Sort By:
Oncogene|April 9, 2008
Forkhead transcription factors and ageingL Partridge, J C Brüning
Best Practice & Research. Clinical Endocrinology & Metabolism|September 10, 2003
Genetic manipulation of the insulin signalling cascade in mice--potential insight into the pathomechanism of type 2 diabetesS Baudler, W Krone, J C Brüning
Deutsche Medizinische Wochenschrift (1946)|May 8, 2009
[The importance of the brain for the regulation of energy and glucose metabolism]E Rother, S D Jordan, J C Brüning
Molecular and Cellular Biology|March 1, 1997
Differential signaling by insulin receptor substrate 1 (IRS-1) and IRS-2 in IRS-1-deficient cellsJ C Brüning, J Winnay, B Cheatham, et al.
Medizinische Klinik (Munich, Germany : 1983)|November 11, 1999
[Conditional mutagenesis--second generation knockout mice as models for internal diseases]J C Brüning, C R Kahn, W Krone, et al.
The Journal of Biological Chemistry|April 1, 2000
Gab-1-mediated IGF-1 signaling in IRS-1-deficient 3T3 fibroblastsJ N Winnay, J C Brüning, D J Burks, et al.
Journal of Pediatric Endocrinology & Metabolism : JPEM|February 24, 2001
Knockout mice challenge our concepts of glucose homeostasis and the pathogenesis of diabetes mellitusC R Kahn, J C Brüning, M D Michael, et al.
Physiology (Bethesda, Md.)|May 13, 2005
Transgenic and knockout mice in diabetes research: novel insights into pathophysiology, limitations, and perspectivesL Plum, F T Wunderlich, S Baudler, et al.
Cell|February 21, 1997
Development of a novel polygenic model of NIDDM in mice heterozygous for IR and IRS-1 null allelesJ C Brüning, J Winnay, S Bonner-Weir, et al.
Cell|February 20, 1999
Tissue-specific knockout of the insulin receptor in pancreatic beta cells creates an insulin secretory defect similar to that in type 2 diabetesR N Kulkarni, J C Brüning, J N Winnay, et al.
Pageof 3

Showing results (1-10 of 21) with videos related to

Sort By:
Pageof 3
Oncogene|April 9, 2008
Forkhead transcription factors and ageingL Partridge, J C Brüning
Best Practice & Research. Clinical Endocrinology & Metabolism|September 10, 2003
Genetic manipulation of the insulin signalling cascade in mice--potential insight into the pathomechanism of type 2 diabetesS Baudler, W Krone, J C Brüning
Deutsche Medizinische Wochenschrift (1946)|May 8, 2009
[The importance of the brain for the regulation of energy and glucose metabolism]E Rother, S D Jordan, J C Brüning
Molecular and Cellular Biology|March 1, 1997
Differential signaling by insulin receptor substrate 1 (IRS-1) and IRS-2 in IRS-1-deficient cellsJ C Brüning, J Winnay, B Cheatham, et al.
Medizinische Klinik (Munich, Germany : 1983)|November 11, 1999
[Conditional mutagenesis--second generation knockout mice as models for internal diseases]J C Brüning, C R Kahn, W Krone, et al.
The Journal of Biological Chemistry|April 1, 2000
Gab-1-mediated IGF-1 signaling in IRS-1-deficient 3T3 fibroblastsJ N Winnay, J C Brüning, D J Burks, et al.
Journal of Pediatric Endocrinology & Metabolism : JPEM|February 24, 2001
Knockout mice challenge our concepts of glucose homeostasis and the pathogenesis of diabetes mellitusC R Kahn, J C Brüning, M D Michael, et al.
Physiology (Bethesda, Md.)|May 13, 2005
Transgenic and knockout mice in diabetes research: novel insights into pathophysiology, limitations, and perspectivesL Plum, F T Wunderlich, S Baudler, et al.
Cell|February 21, 1997
Development of a novel polygenic model of NIDDM in mice heterozygous for IR and IRS-1 null allelesJ C Brüning, J Winnay, S Bonner-Weir, et al.
Cell|February 20, 1999
Tissue-specific knockout of the insulin receptor in pancreatic beta cells creates an insulin secretory defect similar to that in type 2 diabetesR N Kulkarni, J C Brüning, J N Winnay, et al.
Pageof 3