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Neuroscience
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April 20, 2001
Transgenic mice with neuronal overexpression of bcl-2 gene present navigation disabilities in a water task
L Rondi-Reig, Y Lemaigre-Dubreuil, C Montécot, et al.
The European Journal of Neuroscience
|
June 26, 1999
Hu-Bcl-2 transgenic mice with supernumerary neurons exhibit timing impairment in a complex motor task
L Rondi-Reig, A Lohof, Y L Dubreuil, et al.
Apoptosis : an International Journal on Programmed Cell Death
|
October 11, 2005
Upstream control of apoptosis by caspase-2 in serum-deprived primary neurons
D Chauvier, H Lecoeur, A Langonné, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience
|
November 1, 1995
Bcl-2 overexpression prevents motoneuron cell body loss but not axonal degeneration in a mouse model of a neurodegenerative disease
Y Sagot, M Dubois-Dauphin, S A Tan, et al.
The Journal of Cell Biology
|
October 1, 1996
Dissecting processing and apoptotic activity of a cysteine protease by mutant analysis
B Allet, A Hochmann, I Martinou, et al.
Development (Cambridge, England)
|
December 1, 1991
Selective expression of an acetylcholine receptor-lacZ transgene in synaptic nuclei of adult muscle fibers
J R Sanes, Y R Johnson, P T Kotzbauer, et al.
Experimental Neurology
|
July 23, 1999
Overproduction of Cu/Zn-superoxide dismutase or Bcl-2 prevents the brain mitochondrial respiratory dysfunction induced by glutathione depletion
M Mérad-Saïdoune, E Boitier, A Nicole, et al.
Molecular Cell
|
October 5, 2001
Phosphorylation of bid by casein kinases I and II regulates its cleavage by caspase 8
S Desagher, A Osen-Sand, S Montessuit, et al.
The Journal of Cell Biology
|
October 8, 1998
Bax-induced cytochrome C release from mitochondria is independent of the permeability transition pore but highly dependent on Mg2+ ions
R Eskes, B Antonsson, A Osen-Sand, et al.
The Journal of Cell Biology
|
March 23, 1999
Bid-induced conformational change of Bax is responsible for mitochondrial cytochrome c release during apoptosis
S Desagher, A Osen-Sand, A Nichols, et al.
Page
of 9
Search research articles
Search
Showing results (61-70 of 86) with videos related to
Sort By:
Page
of 9
Neuroscience
|
April 20, 2001
Transgenic mice with neuronal overexpression of bcl-2 gene present navigation disabilities in a water task
L Rondi-Reig, Y Lemaigre-Dubreuil, C Montécot, et al.
The European Journal of Neuroscience
|
June 26, 1999
Hu-Bcl-2 transgenic mice with supernumerary neurons exhibit timing impairment in a complex motor task
L Rondi-Reig, A Lohof, Y L Dubreuil, et al.
Apoptosis : an International Journal on Programmed Cell Death
|
October 11, 2005
Upstream control of apoptosis by caspase-2 in serum-deprived primary neurons
D Chauvier, H Lecoeur, A Langonné, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience
|
November 1, 1995
Bcl-2 overexpression prevents motoneuron cell body loss but not axonal degeneration in a mouse model of a neurodegenerative disease
Y Sagot, M Dubois-Dauphin, S A Tan, et al.
The Journal of Cell Biology
|
October 1, 1996
Dissecting processing and apoptotic activity of a cysteine protease by mutant analysis
B Allet, A Hochmann, I Martinou, et al.
Development (Cambridge, England)
|
December 1, 1991
Selective expression of an acetylcholine receptor-lacZ transgene in synaptic nuclei of adult muscle fibers
J R Sanes, Y R Johnson, P T Kotzbauer, et al.
Experimental Neurology
|
July 23, 1999
Overproduction of Cu/Zn-superoxide dismutase or Bcl-2 prevents the brain mitochondrial respiratory dysfunction induced by glutathione depletion
M Mérad-Saïdoune, E Boitier, A Nicole, et al.
Molecular Cell
|
October 5, 2001
Phosphorylation of bid by casein kinases I and II regulates its cleavage by caspase 8
S Desagher, A Osen-Sand, S Montessuit, et al.
The Journal of Cell Biology
|
October 8, 1998
Bax-induced cytochrome C release from mitochondria is independent of the permeability transition pore but highly dependent on Mg2+ ions
R Eskes, B Antonsson, A Osen-Sand, et al.
The Journal of Cell Biology
|
March 23, 1999
Bid-induced conformational change of Bax is responsible for mitochondrial cytochrome c release during apoptosis
S Desagher, A Osen-Sand, A Nichols, et al.
Page
of 9