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J Christopher States

Showing results (31-40 of 96) with videos related to

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Biochemistry|June 21, 2006
Robust incision of Benoz[a]pyrene-7,8-dihyrodiol-9,10-epoxide-DNA adducts by a recombinant thermoresistant interspecies combination UvrABC endonuclease systemGuo Hui Jiang, Milan Skorvaga, Deborah L Croteau, et al.
Toxicology and Applied Pharmacology|October 22, 2023
Clonal variability in chromosomal instability as a potential driver in the acquisition of tumorigenic phenotype in chronic arsenic-exposed and hsa-miR-186 overexpressing human keratinocytesAngeliki Lykoudi, Ana P Ferragut Cardoso, Sandra S Wise, et al.
Seminars in Cancer Biology|May 12, 2021
miRNA dysregulation is an emerging modulator of genomic instabilityAna P Ferragut Cardoso, Mayukh Banerjee, Alexandra N Nail, et al.
Pharmacogenetics|July 1, 2004
Identification of the major promoter and non-coding exons of the human arylamine N-acetyltransferase 1 gene (NAT1)Anwar Husain, David F Barker, J Christopher States, et al.
Biochemistry and Biophysics Reports|August 27, 2015
Folate-Dependent Hydrolysis of Acetyl-Coenzyme A by Recombinant Human and Rodent Arylamine N-AcetyltransferasesMarcus W Stepp, Galina Mamaliga, Mark A Doll, et al.
Drug Metabolism and Disposition: the Biological Fate of Chemicals|October 20, 2011
Phenotype of the most common "slow acetylator" arylamine N-acetyltransferase 1 genetic variant (NAT1*14B) is substrate-dependentLori M Millner, Mark A Doll, Jian Cai, et al.
DNA Repair|September 6, 2005
Telomerase-immortalized human fibroblasts retain UV-induced mutagenesis and p53-mediated DNA damage responsesPaul C Porter, Denise R Clark, Lisa D McDaniel, et al.
Carcinogenesis|November 25, 2011
Functional analysis of arylamine N-acetyltransferase 1 (NAT1) NAT1*10 haplotypes in a complete NATb mRNA constructLori M Millner, Mark A Doll, Marcus W Stepp, et al.
Ecotoxicology and Environmental Safety|March 29, 2023
miR-186 induces tetraploidy in arsenic exposed human keratinocytesAna P Ferragut Cardoso, Alexandra N Nail, Mayukh Banerjee, et al.
Environmental Toxicology and Pharmacology|February 25, 2024
Chronic arsenic exposure suppresses proteasomal and autophagic protein degradationIsabell I Augenstein, Alexandra N Nail, Ana P Ferragut Cardoso, et al.
Pageof 10

Showing results (31-40 of 96) with videos related to

Sort By:
Pageof 10
Biochemistry|June 21, 2006
Robust incision of Benoz[a]pyrene-7,8-dihyrodiol-9,10-epoxide-DNA adducts by a recombinant thermoresistant interspecies combination UvrABC endonuclease systemGuo Hui Jiang, Milan Skorvaga, Deborah L Croteau, et al.
Toxicology and Applied Pharmacology|October 22, 2023
Clonal variability in chromosomal instability as a potential driver in the acquisition of tumorigenic phenotype in chronic arsenic-exposed and hsa-miR-186 overexpressing human keratinocytesAngeliki Lykoudi, Ana P Ferragut Cardoso, Sandra S Wise, et al.
Seminars in Cancer Biology|May 12, 2021
miRNA dysregulation is an emerging modulator of genomic instabilityAna P Ferragut Cardoso, Mayukh Banerjee, Alexandra N Nail, et al.
Pharmacogenetics|July 1, 2004
Identification of the major promoter and non-coding exons of the human arylamine N-acetyltransferase 1 gene (NAT1)Anwar Husain, David F Barker, J Christopher States, et al.
Biochemistry and Biophysics Reports|August 27, 2015
Folate-Dependent Hydrolysis of Acetyl-Coenzyme A by Recombinant Human and Rodent Arylamine N-AcetyltransferasesMarcus W Stepp, Galina Mamaliga, Mark A Doll, et al.
Drug Metabolism and Disposition: the Biological Fate of Chemicals|October 20, 2011
Phenotype of the most common "slow acetylator" arylamine N-acetyltransferase 1 genetic variant (NAT1*14B) is substrate-dependentLori M Millner, Mark A Doll, Jian Cai, et al.
DNA Repair|September 6, 2005
Telomerase-immortalized human fibroblasts retain UV-induced mutagenesis and p53-mediated DNA damage responsesPaul C Porter, Denise R Clark, Lisa D McDaniel, et al.
Carcinogenesis|November 25, 2011
Functional analysis of arylamine N-acetyltransferase 1 (NAT1) NAT1*10 haplotypes in a complete NATb mRNA constructLori M Millner, Mark A Doll, Marcus W Stepp, et al.
Ecotoxicology and Environmental Safety|March 29, 2023
miR-186 induces tetraploidy in arsenic exposed human keratinocytesAna P Ferragut Cardoso, Alexandra N Nail, Mayukh Banerjee, et al.
Environmental Toxicology and Pharmacology|February 25, 2024
Chronic arsenic exposure suppresses proteasomal and autophagic protein degradationIsabell I Augenstein, Alexandra N Nail, Ana P Ferragut Cardoso, et al.
Pageof 10