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Showing results (61-70 of 96) with videos related to

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Environmental and Molecular Mutagenesis|September 7, 2019
Role of Human N-Acetyltransferase 2 Genetic Polymorphism on Aromatic Amine Carcinogen-Induced DNA Damage and Mutagenicity in a Chinese Hamster Ovary Cell Mutation AssayKristin J Baldauf, Raúl A Salazar-González, Mark A Doll, et al.
BMC Cancer|April 1, 2017
Congenic rats with higher arylamine N-acetyltransferase 2 activity exhibit greater carcinogen-induced mammary tumor susceptibility independent of carcinogen metabolismMarcus W Stepp, Mark A Doll, David J Samuelson, et al.
Chemical Research in Toxicology|March 18, 2004
Supercoiled DNA promotes formation of intercalated cis-N2-deoxyguanine adducts and base-stacked trans-N2-deoxyguanine adducts by (+)-7R,8S-dihydrodiol-9S,10R-epoxy-7,8,9,10-tetra- hydrobenzo[a]pyreneGuohui Jiang, Ryszard Jankowiak, Nenad Grubor, et al.
Toxicology and Applied Pharmacology|June 12, 2025
DNA damage response inhibition is an early event in cadmium-induced breast carcinogenesisAlexandra N Nail, Ashley V Chavez, Aliyah N Bailey, et al.
Toxicology and Applied Pharmacology|August 25, 2010
Evaluation of the serum catalase and myeloperoxidase activities in chronic arsenic-exposed individuals and concomitant cytogenetic damageMayukh Banerjee, Nilanjana Banerjee, Pritha Ghosh, et al.
Mutation Research|November 2, 2010
Precancerous and non-cancer disease endpoints of chronic arsenic exposure: the level of chromosomal damage and XRCC3 T241M polymorphismManjari Kundu, Pritha Ghosh, Sanhita Mitra, et al.
The Journal of Pharmacology and Experimental Therapeutics|January 18, 2018
High N-Acetyltransferase 1 Expression Is Associated with Estrogen Receptor Expression in Breast Tumors, but Is not Under Direct Regulation by Estradiol, 5<i>α</i>-androstane-3<i>β</i>,17<i>β</i>-Diol, or Dihydrotestosterone in Breast Cancer CellsXiaoyan Zhang, Samantha M Carlisle, Mark A Doll, et al.
Archives of Toxicology|May 25, 2021
Dynamic alteration in miRNA and mRNA expression profiles at different stages of chronic arsenic exposure-induced carcinogenesis in a human cell culture model of skin cancerMayukh Banerjee, Ana Ferragut Cardoso, Laila Al-Eryani, et al.
Toxicology and Applied Pharmacology|March 11, 2008
Sensitivity to sodium arsenite in human melanoma cells depends upon susceptibility to arsenite-induced mitotic arrestSamuel C McNeely, Alex C Belshoff, B Frazier Taylor, et al.
Cancer Epidemiology, Biomarkers & Prevention : a Publication of the American Association for Cancer Research, Cosponsored by the American Society of Preventive Oncology|July 14, 2007
2-Amino-3,8-dimethylimidazo-[4,5-f]quinoxaline-induced DNA adduct formation and mutagenesis in DNA repair-deficient Chinese hamster ovary cells expressing human cytochrome P4501A1 and rapid or slow acetylator N-acetyltransferase 2Jean Bendaly, Shuang Zhao, Jason R Neale, et al.
Pageof 10

Showing results (61-70 of 96) with videos related to

Sort By:
Pageof 10
Environmental and Molecular Mutagenesis|September 7, 2019
Role of Human N-Acetyltransferase 2 Genetic Polymorphism on Aromatic Amine Carcinogen-Induced DNA Damage and Mutagenicity in a Chinese Hamster Ovary Cell Mutation AssayKristin J Baldauf, Raúl A Salazar-González, Mark A Doll, et al.
BMC Cancer|April 1, 2017
Congenic rats with higher arylamine N-acetyltransferase 2 activity exhibit greater carcinogen-induced mammary tumor susceptibility independent of carcinogen metabolismMarcus W Stepp, Mark A Doll, David J Samuelson, et al.
Chemical Research in Toxicology|March 18, 2004
Supercoiled DNA promotes formation of intercalated cis-N2-deoxyguanine adducts and base-stacked trans-N2-deoxyguanine adducts by (+)-7R,8S-dihydrodiol-9S,10R-epoxy-7,8,9,10-tetra- hydrobenzo[a]pyreneGuohui Jiang, Ryszard Jankowiak, Nenad Grubor, et al.
Toxicology and Applied Pharmacology|June 12, 2025
DNA damage response inhibition is an early event in cadmium-induced breast carcinogenesisAlexandra N Nail, Ashley V Chavez, Aliyah N Bailey, et al.
Toxicology and Applied Pharmacology|August 25, 2010
Evaluation of the serum catalase and myeloperoxidase activities in chronic arsenic-exposed individuals and concomitant cytogenetic damageMayukh Banerjee, Nilanjana Banerjee, Pritha Ghosh, et al.
Mutation Research|November 2, 2010
Precancerous and non-cancer disease endpoints of chronic arsenic exposure: the level of chromosomal damage and XRCC3 T241M polymorphismManjari Kundu, Pritha Ghosh, Sanhita Mitra, et al.
The Journal of Pharmacology and Experimental Therapeutics|January 18, 2018
High N-Acetyltransferase 1 Expression Is Associated with Estrogen Receptor Expression in Breast Tumors, but Is not Under Direct Regulation by Estradiol, 5<i>α</i>-androstane-3<i>β</i>,17<i>β</i>-Diol, or Dihydrotestosterone in Breast Cancer CellsXiaoyan Zhang, Samantha M Carlisle, Mark A Doll, et al.
Archives of Toxicology|May 25, 2021
Dynamic alteration in miRNA and mRNA expression profiles at different stages of chronic arsenic exposure-induced carcinogenesis in a human cell culture model of skin cancerMayukh Banerjee, Ana Ferragut Cardoso, Laila Al-Eryani, et al.
Toxicology and Applied Pharmacology|March 11, 2008
Sensitivity to sodium arsenite in human melanoma cells depends upon susceptibility to arsenite-induced mitotic arrestSamuel C McNeely, Alex C Belshoff, B Frazier Taylor, et al.
Cancer Epidemiology, Biomarkers & Prevention : a Publication of the American Association for Cancer Research, Cosponsored by the American Society of Preventive Oncology|July 14, 2007
2-Amino-3,8-dimethylimidazo-[4,5-f]quinoxaline-induced DNA adduct formation and mutagenesis in DNA repair-deficient Chinese hamster ovary cells expressing human cytochrome P4501A1 and rapid or slow acetylator N-acetyltransferase 2Jean Bendaly, Shuang Zhao, Jason R Neale, et al.
Pageof 10