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Blood|May 15, 1996
Activated abl oncogenes and apoptosis: differing responses of transformed myeloid progenitor cell linesJ Zhu, P M Nabissa, B Hoffman, et al.International Journal of Oncology|June 19, 1998
Role of a mutant p53 protein in apoptosis: characterization of a function independent of transcriptional trans-activationC Guillouf, F Rosselli, R T Sjin, et al.Annals of the New York Academy of Sciences|December 1, 1992
Genotoxic-stress-response genes and growth-arrest genes. gadd, MyD, and other genes induced by treatments eliciting growth arrestA J Fornace, J Jackman, M C Hollander, et al.Molecular and Cellular Biology|April 1, 1994
The novel primary response gene MyD118 and the proto-oncogenes myb, myc, and bcl-2 modulate transforming growth factor beta 1-induced apoptosis of myeloid leukemia cellsM Selvakumaran, H K Lin, R T Sjin, et al.Blood|May 15, 1995
Dissection of the genetic programs of p53-mediated G1 growth arrest and apoptosis: blocking p53-induced apoptosis unmasks G1 arrestC Guillouf, X Graña, M Selvakumaran, et al.Oncogene|November 1, 1994
Induction of p21 (WAF-1/CIP1) during differentiationR A Steinman, B Hoffman, A Iro, et al.Oncogene|September 22, 1999
CR6: A third member in the MyD118 and Gadd45 gene family which functions in negative growth controlW Zhang, I Bae, K Krishnaraju, et al.Molecular and Cellular Biology|September 1, 1991
Leukemia inhibitory factor and interleukin-6 trigger the same immediate early response, including tyrosine phosphorylation, upon induction of myeloid leukemia differentiationK A Lord, A Abdollahi, S M Thomas, et al.Oncogene|June 1, 1994
Tumor suppressor p53 is a regulator of bcl-2 and bax gene expression in vitro and in vivoT Miyashita, S Krajewski, M Krajewska, et al.Pageof 56