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Oncogene|September 12, 2012
Oncogenic KRAS-induced epiregulin overexpression contributes to aggressive phenotype and is a promising therapeutic target in non-small-cell lung cancerN Sunaga, K Kaira, H Imai, et al.
Oncogene|December 8, 2009
Genomic and functional analysis identifies CRKL as an oncogene amplified in lung cancerY H Kim, K A Kwei, L Girard, et al.
Oncogene|November 30, 2005
ASPP1, a common activator of TP53, is inactivated by aberrant methylation of its promoter in acute lymphoblastic leukemiaX Agirre, J Román-Gómez, A Jiménez-Velasco, et al.
Proceedings of the National Academy of Sciences of the United States of America|June 24, 1998
Computerized polymorphic marker identification: experimental validation and a predicted human polymorphism catalogJ W Fondon, G M Mele, R I Brezinschek, et al.
Molecular Cancer Therapeutics|December 7, 2002
DNA methylation profiles of lung tumorsS Toyooka, K O Toyooka, R Maruyama, et al.
Cancer Research|November 3, 2001
Inactivation of human SRBC, located within the 11p15.5-p15.4 tumor suppressor region, in breast and lung cancersX L Xu, L C Wu, F Du, et al.
Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|July 13, 2001
Aberrant methylation of the adenomatous polyposis coli (APC) gene promoter 1A in breast and lung carcinomasA K Virmani, A Rathi, U G Sathyanarayana, et al.
Cancer Research|June 1, 1997
FHIT and FRA3B 3p14.2 allele loss are common in lung cancer and preneoplastic bronchial lesions and are associated with cancer-related FHIT cDNA splicing aberrationsK M Fong, E J Biesterveld, A Virmani, et al.
Cancer Research|August 12, 1998
Characterization of a breast cancer cell line derived from a germ-line BRCA1 mutation carrierG E Tomlinson, T T Chen, V A Stastny, et al.
Oncogene|October 24, 2006
c-Jun N-terminal kinase is activated in non-small-cell lung cancer and promotes neoplastic transformation in human bronchial epithelial cellsT S Khatlani, M Wislez, M Sun, et al.
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