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The Journal of Clinical Investigation|March 1, 1995
The low density lipoprotein receptor is not required for normal catabolism of Lp(a) in humansD J Rader, W A Mann, W Cain, et al.
Transactions of the Association of American Physicians|January 1, 1985
On the circulating form of atrial natriuretic factorM Cantin, J Genest, G Thibault, et al.
Hypertension (Dallas, Tex. : 1979)|June 1, 1989
Atrial natriuretic factor in Purkinje fibers of rabbit heartM B Anand-Srivastava, G Thibault, C Sola, et al.
Journal of Hypertension. Supplement : Official Journal of the International Society of Hypertension|June 1, 1986
Cyclic GMP as mediator and biological marker of atrial natriuretic factorP Hamet, J Tremblay, S C Pang, et al.
The Canadian Journal of Cardiology|February 2, 2006
Rationale, design and baseline characteristics of a large, simple, randomized trial of combined folic acid and vitamins B6 and B12 in high-risk patients: the Heart Outcomes Prevention Evaluation (HOPE)-2 trialE Lonn, C Held, J M O Arnold, et al.
Atherosclerosis|June 28, 2001
Efficacy and short-term safety of a new ACAT inhibitor, avasimibe, on lipids, lipoproteins, and apolipoproteins, in patients with combined hyperlipidemiaW Insull, M Koren, J Davignon, et al.
The Journal of Biological Chemistry|January 25, 1992
Two naturally occurring mutations at the first and second bases of codon aspartic acid 156 in the proposed catalytic triad of human lipoprotein lipase. In vivo evidence that aspartic acid 156 is essential for catalysisY H Ma, T Bruin, S Tuzgol, et al.
Proceedings of the National Academy of Sciences of the United States of America|May 1, 1984
Amino acid sequence of homologous rat atrial peptides: natriuretic activity of native and synthetic formsN G Seidah, C Lazure, M Chrétien, et al.
Atherosclerosis|March 11, 1991
Restriction fragment length polymorphisms of the apolipoprotein A-I, C-III, A-IV gene locus. Relationships with lipids, apolipoproteins, and premature coronary artery diseaseJ M Ordovas, F Civeira, J Genest, et al.
Journal of Lipid Research|August 1, 1990
Familial defective apolipoprotein B-100: a mutation of apolipoprotein B that causes hypercholesterolemiaT L Innerarity, R W Mahley, K H Weisgraber, et al.
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