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The Journal of Biological Chemistry|January 25, 1992
Two naturally occurring mutations at the first and second bases of codon aspartic acid 156 in the proposed catalytic triad of human lipoprotein lipase. In vivo evidence that aspartic acid 156 is essential for catalysisY H Ma, T Bruin, S Tuzgol, et al.Journal of Lipid Research|August 1, 1990
Familial defective apolipoprotein B-100: a mutation of apolipoprotein B that causes hypercholesterolemiaT L Innerarity, R W Mahley, K H Weisgraber, et al.Clinical Chemistry|November 2, 1999
Ratio of remnant-like particle-cholesterol to serum total triglycerides is an effective alternative to ultracentrifugal and electrophoretic methods in the diagnosis of familial type III hyperlipoproteinemiaT Wang, K Nakajima, E T Leary, et al.Arteriosclerosis, Thrombosis, and Vascular Biology|November 5, 1997
Efficacy and safety of a new hydroxymethylglutaryl-coenzyme A reductase inhibitor, atorvastatin, in patients with combined hyperlipidemia: comparison with fenofibrateT C Ooi, T Heinonen, P Alaupovic, et al.Atherosclerosis|November 2, 2001
Cholesterol and apolipoprotein B metabolism in Tangier diseaseE J Schaefer, M E Brousseau, M R Diffenderfer, et al.Journal of Medical Genetics|August 19, 2008
A PCSK9 variant and familial combined hyperlipidaemiaM Abifadel, L Bernier, G Dubuc, et al.JAMA|January 10, 1996
Efficacy and safety of a new HMG-CoA reductase inhibitor, atorvastatin, in patients with hypertriglyceridemiaR G Bakker-Arkema, M H Davidson, R J Goldstein, et al.Clinical Genetics|July 1, 1997
Geographic distribution of French-Canadian low-density lipoprotein receptor gene mutations in the Province of QuebecM C Vohl, S Moorjani, M Roy, et al.Pageof 22