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Genome Research|August 16, 2020
Somatic structural variation targets neurodevelopmental genes and identifies SHANK2 as a tumor suppressor in neuroblastomaGonzalo Lopez, Karina L Conkrite, Miriam Doepner, et al.
Cancer Research|April 16, 2020
Telomere Maintenance Mechanisms Define Clinical Outcome in High-Risk NeuroblastomaBalakrishna Koneru, Gonzalo Lopez, Ahsan Farooqi, et al.
Cancer Research|August 16, 2023
Integrative Genomic Analyses Identify LncRNA Regulatory Networks across Pediatric Leukemias and Solid TumorsApexa Modi, Gonzalo Lopez, Karina L Conkrite, et al.
Cell Metabolism|September 22, 2015
MYC Disrupts the Circadian Clock and Metabolism in Cancer CellsBrian J Altman, Annie L Hsieh, Arjun Sengupta, et al.
Neuro-Oncology|July 2, 2020
European genetic ancestry associated with risk of childhood ependymomaChenan Zhang, Quinn T Ostrom, Helen M Hansen, et al.
Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|October 9, 2023
Surface and Global Proteome Analyses Identify ENPP1 and Other Surface Proteins as Actionable Immunotherapeutic Targets in Ewing SarcomaBrian Mooney, Gian Luca Negri, Taras Shyp, et al.
Nature Genetics|May 5, 2009
Common variations in BARD1 influence susceptibility to high-risk neuroblastomaMario Capasso, Marcella Devoto, Cuiping Hou, et al.
Cancer Research|February 22, 2012
Common variation at BARD1 results in the expression of an oncogenic isoform that influences neuroblastoma susceptibility and oncogenicityKristopher R Bosse, Sharon J Diskin, Kristina A Cole, et al.
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