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Nature|August 23, 1990
Suppression of c-ras transformation by GTPase-activating proteinK Zhang, J E DeClue, W C Vass, et al.
Proceedings of the National Academy of Sciences of the United States of America|August 20, 1996
Suppression of tumorigenicity by the wild-type tuberous sclerosis 2 (Tsc2) gene and its C-terminal regionF Jin, R Wienecke, G H Xiao, et al.
Annals of Neurology|August 1, 1997
Reduced TSC2 RNA and protein in sporadic astrocytomas and ependymomasR Wienecke, A Guha, J C Maize, et al.
The American Journal of Pathology|January 1, 1997
Expression of the TSC2 product tuberin and its target Rap1 in normal human tissuesR Wienecke, J C Maize, J A Reed, et al.
Molecular and Cellular Biology|June 22, 1999
Ras-specific exchange factor GRF: oligomerization through its Dbl homology domain and calcium-dependent activation of RafP H Anborgh, X Qian, A G Papageorge, et al.
Molecular and Cellular Biology|January 1, 1994
Neurofibromin can inhibit Ras-dependent growth by a mechanism independent of its GTPase-accelerating functionM R Johnson, J E DeClue, S Felzmann, et al.
The Journal of Biological Chemistry|January 24, 1997
Sequential modification of serines 621 and 624 in the Raf-1 carboxyl terminus produces alterations in its electrophoretic mobilityA F Ferrier, M Lee, W B Anderson, et al.
The Journal of Clinical Investigation|May 3, 2000
Epidermal growth factor receptor expression in neurofibromatosis type 1-related tumors and NF1 animal modelsJ E DeClue, S Heffelfinger, G Benvenuto, et al.
Molecular and Cellular Biology|June 1, 1991
A ras effector domain mutant which is temperature sensitive for cellular transformation: interactions with GTPase-activating protein and NF-1J E DeClue, J C Stone, R A Blanchard, et al.
Human Molecular Genetics|December 14, 2001
Pathological mutations in TSC1 and TSC2 disrupt the interaction between hamartin and tuberinA K Hodges, S Li, J Maynard, et al.
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