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J E Maggio

Showing results (61-70 of 71) with videos related to

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Peptides|November 1, 1988
Substance P and substance K receptor binding sites in the human gastrointestinal tract: localization by autoradiographyT S Gates, R P Zimmerman, C R Mantyh, et al.
Brain Research|February 12, 1999
Delayed behavioral effects following intrahippocampal injection of aggregated A beta (1-42)E O'Hare, D T Weldon, P W Mantyh, et al.
Proceedings of the National Academy of Sciences of the United States of America|May 1, 1988
Receptor binding sites for substance P, but not substance K or neuromedin K, are expressed in high concentrations by arterioles, venules, and lymph nodules in surgical specimens obtained from patients with ulcerative colitis and Crohn diseaseC R Mantyh, T S Gates, R P Zimmerman, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|April 18, 1998
Fibrillar beta-amyloid induces microglial phagocytosis, expression of inducible nitric oxide synthase, and loss of a select population of neurons in the rat CNS in vivoD T Weldon, S D Rogers, J R Ghilardi, et al.
Journal of Structural Biology|August 15, 2000
The Alzheimer's peptide a beta adopts a collapsed coil structure in waterS Zhang, K Iwata, M J Lachenmann, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 28, 1995
Rapid endocytosis of a G protein-coupled receptor: substance P evoked internalization of its receptor in the rat striatum in vivoP W Mantyh, C J Allen, J R Ghilardi, et al.
Science (New York, N.Y.)|June 16, 1995
Receptor endocytosis and dendrite reshaping in spinal neurons after somatosensory stimulationP W Mantyh, E DeMaster, A Malhotra, et al.
Biochemistry|November 5, 1996
Point substitution in the central hydrophobic cluster of a human beta-amyloid congener disrupts peptide folding and abolishes plaque competenceW P Esler, E R Stimson, J R Ghilardi, et al.
Biochemistry|August 11, 1999
Deposition of monomeric, not oligomeric, Abeta mediates growth of Alzheimer's disease amyloid plaques in human brain preparationsB P Tseng, W P Esler, C B Clish, et al.
Journal of Structural Biology|August 15, 2000
Activation barriers to structural transition determine deposition rates of Alzheimer's disease a beta amyloidW P Esler, A M Felix, E R Stimson, et al.
Pageof 8

Showing results (61-70 of 71) with videos related to

Sort By:
Pageof 8
Peptides|November 1, 1988
Substance P and substance K receptor binding sites in the human gastrointestinal tract: localization by autoradiographyT S Gates, R P Zimmerman, C R Mantyh, et al.
Brain Research|February 12, 1999
Delayed behavioral effects following intrahippocampal injection of aggregated A beta (1-42)E O'Hare, D T Weldon, P W Mantyh, et al.
Proceedings of the National Academy of Sciences of the United States of America|May 1, 1988
Receptor binding sites for substance P, but not substance K or neuromedin K, are expressed in high concentrations by arterioles, venules, and lymph nodules in surgical specimens obtained from patients with ulcerative colitis and Crohn diseaseC R Mantyh, T S Gates, R P Zimmerman, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|April 18, 1998
Fibrillar beta-amyloid induces microglial phagocytosis, expression of inducible nitric oxide synthase, and loss of a select population of neurons in the rat CNS in vivoD T Weldon, S D Rogers, J R Ghilardi, et al.
Journal of Structural Biology|August 15, 2000
The Alzheimer's peptide a beta adopts a collapsed coil structure in waterS Zhang, K Iwata, M J Lachenmann, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 28, 1995
Rapid endocytosis of a G protein-coupled receptor: substance P evoked internalization of its receptor in the rat striatum in vivoP W Mantyh, C J Allen, J R Ghilardi, et al.
Science (New York, N.Y.)|June 16, 1995
Receptor endocytosis and dendrite reshaping in spinal neurons after somatosensory stimulationP W Mantyh, E DeMaster, A Malhotra, et al.
Biochemistry|November 5, 1996
Point substitution in the central hydrophobic cluster of a human beta-amyloid congener disrupts peptide folding and abolishes plaque competenceW P Esler, E R Stimson, J R Ghilardi, et al.
Biochemistry|August 11, 1999
Deposition of monomeric, not oligomeric, Abeta mediates growth of Alzheimer's disease amyloid plaques in human brain preparationsB P Tseng, W P Esler, C B Clish, et al.
Journal of Structural Biology|August 15, 2000
Activation barriers to structural transition determine deposition rates of Alzheimer's disease a beta amyloidW P Esler, A M Felix, E R Stimson, et al.
Pageof 8