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Journal of Immunology (Baltimore, Md. : 1950)|November 1, 1983
Studies of the mechanism of bacterial resistance to complement-mediated killing. V. IgG and F(ab')2 mediate killing of E. coli 0111B4 by the alternative complement pathway without increasing C5b-9 depositionK A Joiner, R C Goldman, C H Hammer, et al.
Infection and Immunity|July 1, 1984
Mechanism of bacterial resistance to complement-mediated killing: inserted C5b-9 correlates with killing for Escherichia coli O111B4 varying in O-antigen capsule and O-polysaccharide coverage of lipid A core oligosaccharideK A Joiner, M A Schmetz, R C Goldman, et al.
The New England Journal of Medicine|July 1, 1976
The glomerular complement receptor in immunologically mediated renal glomerular injuryM C Gelfand, M L Shin, R B Nagle, et al.
Arthritis and Rheumatism|January 1, 1982
A serial study of splenic reticuloendothelial system Fc receptor functional activity in systemic lupus erythematosusM I Hamburger, T J Lawley, R P Kimberly, et al.
British Journal of Haematology|August 1, 1986
Serum sickness and haematopoietic recovery with antithymocyte globulin in bone marrow failure patientsL Bielory, P Gascon, T J Lawley, et al.
Clinical Immunology and Immunopathology|October 1, 1993
Increased expression of Fc gamma RI on isolated PMN from individuals of African descentM M Moxey-Mims, M M Frank, E Y Lin, et al.
Lancet (London, England)|April 11, 1981
Impaired immune response of splenectomised patients to polyvalent pneumococcal vaccineS W Hosea, C G Burch, E J Brown, et al.
The Journal of Infectious Diseases|January 1, 1975
Serum complement and immunity in experimental simian malaria. I. Cyclical alterations in C4 related to schizont ruptureR H Glew, J P Atkinson, M M Frank, et al.
The Journal of Clinical Investigation|March 1, 1987
Demonstration of defective C3-receptor-mediated clearance by the reticuloendothelial system in patients with acquired immunodeficiency syndromeB S Bender, J F Bohnsack, S H Sourlis, et al.
The Journal of Experimental Medicine|March 1, 1982
Studies on the mechanism of bacterial resistance to complement-mediated killing. I. Terminal complement components are deposited and released from Salmonella minnesota S218 without causing bacterial deathK A Joiner, C H Hammer, E J Brown, et al.
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