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Neurobiology of Disease|October 21, 1999
Mutant huntingtin forms in vivo complexes with distinct context-dependent conformations of the polyglutamine segmentF Persichetti, F Trettel, C C Huang, et al.
Current Opinion in Neurobiology|October 1, 1996
Huntington's disease: translating a CAG repeat into a pathogenic mechanismM E MacDonald, J F Gusella
Nature Reviews. Neuroscience|March 17, 2001
Molecular genetics: unmasking polyglutamine triggers in neurodegenerative diseaseJ F Gusella, M E MacDonald
Seminars in Cell Biology|February 1, 1995
Huntington's diseaseJ F Gusella, M E MacDonald
Annual Review of Medicine|January 1, 1996
Trinucleotide instability: a repeating theme in human inherited disordersJ F Gusella, M E MacDonald
Current Opinion in Neurobiology|October 1, 1995
Huntington's disease: CAG genetics expands neurobiologyJ F Gusella, M E MacDonald
Current Opinion in Neurobiology|August 4, 1998
Huntingtin: a single bait hooks many speciesJ F Gusella, M E MacDonald
Somatic Cell and Molecular Genetics|July 20, 1999
Amyloid formation by mutant huntingtin: threshold, progressivity and recruitment of normal polyglutamine proteinsC C Huang, P W Faber, F Persichetti, et al.
Somatic Cell and Molecular Genetics|March 1, 1994
Mouse Huntington's disease gene homolog (Hdh)G T Barnes, M P Duyao, C M Ambrose, et al.
Human Molecular Genetics|January 1, 1994
Analysis of the trinucleotide repeat expansion in Italian families affected with Huntington diseaseA Novelletto, F Persichetti, G Sabbadini, et al.
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