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J F Terdiman

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Computers and Biomedical Research, an International Journal|October 5, 1970
Mass random storage devices and their application to a Medical Information System (MIS)J F Terdiman
Annual Review of Biophysics and Bioengineering|January 1, 1973
Technology of multiphasic patient screeningM F Collen, J F Terdiman
Federation Proceedings|December 1, 1974
Functional goals and problems in large-scale patient record management and automated screeningM F Collen, L S Davis, E E van Brunt, et al.
The Journal of Clinical Investigation|March 1, 1990
Familial hypobetalipoproteinemia caused by a mutation in the apolipoprotein B gene that results in a truncated species of apolipoprotein B (B-31). A unique mutation that helps to define the portion of the apolipoprotein B molecule required for the formation of buoyant, triglyceride-rich lipoproteinsS G Young, S T Hubl, R S Smith, et al.
Methods of Information in Medicine|January 5, 2011
Current status of a medical information systemE E van Brunt, L S Davis, J F Terdiman, et al.
The New England Journal of Medicine|June 15, 1989
Familial hypobetalipoproteinemia associated with a mutant species of apolipoprotein B (B-46)S G Young, S T Hubl, D A Chappell, et al.
The American Journal of Medicine|November 1, 1993
Relationship of apolipoprotein E phenotypes to hypocholesterolemiaS M Synder, J F Terdiman, B Caan, et al.
Journal of Lipid Research|March 1, 1993
Four new mutations in the apolipoprotein B gene causing hypobetalipoproteinemia, including two different frameshift mutations that yield truncated apolipoprotein B proteins of identical lengthS G Young, C R Pullinger, B R Zysow, et al.
Pageof 1

Showing results (1-10 of 8) with videos related to

Sort By:
Pageof 1
Computers and Biomedical Research, an International Journal|October 5, 1970
Mass random storage devices and their application to a Medical Information System (MIS)J F Terdiman
Annual Review of Biophysics and Bioengineering|January 1, 1973
Technology of multiphasic patient screeningM F Collen, J F Terdiman
Federation Proceedings|December 1, 1974
Functional goals and problems in large-scale patient record management and automated screeningM F Collen, L S Davis, E E van Brunt, et al.
The Journal of Clinical Investigation|March 1, 1990
Familial hypobetalipoproteinemia caused by a mutation in the apolipoprotein B gene that results in a truncated species of apolipoprotein B (B-31). A unique mutation that helps to define the portion of the apolipoprotein B molecule required for the formation of buoyant, triglyceride-rich lipoproteinsS G Young, S T Hubl, R S Smith, et al.
Methods of Information in Medicine|January 5, 2011
Current status of a medical information systemE E van Brunt, L S Davis, J F Terdiman, et al.
The New England Journal of Medicine|June 15, 1989
Familial hypobetalipoproteinemia associated with a mutant species of apolipoprotein B (B-46)S G Young, S T Hubl, D A Chappell, et al.
The American Journal of Medicine|November 1, 1993
Relationship of apolipoprotein E phenotypes to hypocholesterolemiaS M Synder, J F Terdiman, B Caan, et al.
Journal of Lipid Research|March 1, 1993
Four new mutations in the apolipoprotein B gene causing hypobetalipoproteinemia, including two different frameshift mutations that yield truncated apolipoprotein B proteins of identical lengthS G Young, C R Pullinger, B R Zysow, et al.
Pageof 1