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The American Journal of Physiology|June 11, 1994
Thapsigargin inhibits the glucose-induced decrease of intracellular Ca2+ in mouse islets of LangerhansM W Roe, R J Mertz, M E Lancaster, et al.The Journal of General Physiology|March 1, 1994
Ion permeation, divalent ion block, and chemical modification of single sodium channels. Description by single- and double-occupancy rate-theory modelsR J French, J F Worley, W F Wonderlin, et al.The Journal of Biological Chemistry|May 15, 1993
Voltage-dependent intracellular calcium release from mouse islets stimulated by glucoseM W Roe, M E Lancaster, R J Mertz, et al.The Journal of Biological Chemistry|May 20, 1994
Endoplasmic reticulum calcium store regulates membrane potential in mouse islet beta-cellsJ F Worley, M S McIntyre, B Spencer, et al.Cancer Research|September 1, 1990
Inhibition of human breast cancer cell proliferation in tissue culture by the neuroleptic agents pimozide and thioridazineJ S Strobl, K L Kirkwood, T K Lantz, et al.The Journal of Biological Chemistry|April 15, 1994
Dependence on NADH produced during glycolysis for beta-cell glucose signalingI D Dukes, M S McIntyre, R J Mertz, et al.The Journal of Biological Chemistry|November 11, 1994
Delayed rectifier K+ channel overexpression in transgenic islets and beta-cells associated with impaired glucose responsivenessL H Philipson, M P Rosenberg, A Kuznetsov, et al.The American Journal of Physiology|January 11, 1996
NIDDM is associated with loss of pancreatic beta-cell L-type Ca2+ channel activityM W Roe, J F Worley, Y Tokuyama, et al.The Journal of Biological Chemistry|July 15, 1994
Defective glucose-dependent endoplasmic reticulum Ca2+ sequestration in diabetic mouse islets of LangerhansM W Roe, L H Philipson, C J Frangakis, et al.The Journal of Biological Chemistry|May 30, 1998
Characterization of a Ca2+ release-activated nonselective cation current regulating membrane potential and [Ca2+]i oscillations in transgenically derived beta-cellsM W Roe, J F Worley, F Qian, et al.Pageof 4