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Nature Medicine|May 10, 2000
Concurrent enteric helminth infection modulates inflammation and gastric immune responses and reduces helicobacter-induced gastric atrophyJ G Fox, P Beck, C A Dangler, et al.Nature Reviews. Immunology|March 22, 2002
Man the barrier! Strategic defences in the intestinal mucosaC Nagler-AndersonCritical Reviews in Immunology|June 29, 2000
Tolerance and immunity in the intestinal immune systemC Nagler-AndersonCurrent Opinion in Gastroenterology|October 7, 2006
Mucosal T-cell responses to enteric infectionH N Shi, C Nagler-AndersonInternational Immunology|January 1, 1990
Resistance of normal, unstimulated, CD8+ T cells to lysis by cytotoxic granules from cloned T cell linesC Nagler-Anderson, H N EisenJournal of Immunology (Baltimore, Md. : 1950)|December 2, 1998
Oral administration of the bacterial superantigen staphylococcal enterotoxin B induces activation and cytokine production by T cells in murine gut-associated lymphoid tissueG M Spiekermann, C Nagler-AndersonCritical Reviews in Immunology|October 20, 2001
Peripheral nonresponsiveness to orally administered soluble protein antigensC Nagler-Anderson, H N ShiJournal of Immunology (Baltimore, Md. : 1950)|October 1, 1992
Self-reactive, T cell receptor-gamma delta+, lymphocytes from the intestinal epithelium of weanling miceC Nagler-Anderson, L A McNair, A CradockJournal of Immunology (Baltimore, Md. : 1950)|November 22, 2000
Enteric infection acts as an adjuvant for the response to a model food antigenH N Shi, H Y Liu, C Nagler-AndersonJournal of Immunology (Baltimore, Md. : 1950)|May 5, 1999
Orally induced peripheral nonresponsiveness is maintained in the absence of functional Th1 or Th2 cellsH N Shi, M J Grusby, C Nagler-AndersonPageof 54