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J Gawler

Showing results (31-40 of 46) with videos related to

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FEBS Letters|August 5, 1985
The phorbol ester TPA prevents the expression of both glucagon desensitisation and the glucagon-mediated block of insulin stimulation of the peripheral plasma membrane cyclic AMP phosphodiesterase in rat hepatocytesC M Heyworth, S P Wilson, D J Gawler, et al.
Neurology|April 24, 1999
Heightened intrathecal release of proinflammatory cytokines in Creutzfeldt-Jakob diseaseM K Sharief, A Green, J P Dick, et al.
BMJ (Clinical Research Ed.)|January 22, 1994
Safe blood? HTLV-1 infection is cripplingJ A Copplestone, A G Prentice, M D Hamon, et al.
The British Journal of Ophthalmology|June 1, 1974
Computer assisted tomography in orbital diseaseJ Gawler, M D Sanders, J W Bull, et al.
Oncogene|March 2, 1995
CaLB: a 43 amino acid calcium-dependent membrane/phospholipid binding domain in p120 Ras GTPase-activating proteinD J Gawler, L J Zhang, M Reedijk, et al.
The Journal of Biological Chemistry|October 4, 1996
The Ca2+-dependent lipid binding domain of P120GAP mediates protein-protein interactions with Ca2+-dependent membrane-binding proteins. Evidence for a direct interaction between annexin VI and P120GAPA J Davis, J T Butt, J H Walker, et al.
Journal of Neurology, Neurosurgery, and Psychiatry|September 1, 1993
A familial disorder associated with palatal myoclonus, other brainstem signs, tetraparesis, ataxia and Rosenthal fibre formationR S Howard, R Greenwood, J Gawler, et al.
Biochemical Society Transactions|February 1, 1987
Glucagon stimulates adenylate cyclase through GR2 glucagon receptors: a process which can be attenuated by glucagon stimulating inositol phospholipid metabolism through GR1 glucagon receptorsM D Houslay, M J Wakelam, G J Murphy, et al.
Journal of Clinical Pathology|August 30, 2005
Guillain Barré syndrome precipitated by the use of antilymphocyte globulin in the treatment of severe aplastic anaemiaB Kaya, C E Davies, H E Oakervee, et al.
The Biochemical Journal|April 1, 1989
Glucagon desensitization of adenylate cyclase and stimulation of inositol phospholipid metabolism does not involve the inhibitory guanine nucleotide regulatory protein Gi, which is inactivated upon challenge of hepatocytes with glucagonG J Murphy, D J Gawler, G Milligan, et al.
Pageof 5

Showing results (31-40 of 46) with videos related to

Sort By:
Pageof 5
FEBS Letters|August 5, 1985
The phorbol ester TPA prevents the expression of both glucagon desensitisation and the glucagon-mediated block of insulin stimulation of the peripheral plasma membrane cyclic AMP phosphodiesterase in rat hepatocytesC M Heyworth, S P Wilson, D J Gawler, et al.
Neurology|April 24, 1999
Heightened intrathecal release of proinflammatory cytokines in Creutzfeldt-Jakob diseaseM K Sharief, A Green, J P Dick, et al.
BMJ (Clinical Research Ed.)|January 22, 1994
Safe blood? HTLV-1 infection is cripplingJ A Copplestone, A G Prentice, M D Hamon, et al.
The British Journal of Ophthalmology|June 1, 1974
Computer assisted tomography in orbital diseaseJ Gawler, M D Sanders, J W Bull, et al.
Oncogene|March 2, 1995
CaLB: a 43 amino acid calcium-dependent membrane/phospholipid binding domain in p120 Ras GTPase-activating proteinD J Gawler, L J Zhang, M Reedijk, et al.
The Journal of Biological Chemistry|October 4, 1996
The Ca2+-dependent lipid binding domain of P120GAP mediates protein-protein interactions with Ca2+-dependent membrane-binding proteins. Evidence for a direct interaction between annexin VI and P120GAPA J Davis, J T Butt, J H Walker, et al.
Journal of Neurology, Neurosurgery, and Psychiatry|September 1, 1993
A familial disorder associated with palatal myoclonus, other brainstem signs, tetraparesis, ataxia and Rosenthal fibre formationR S Howard, R Greenwood, J Gawler, et al.
Biochemical Society Transactions|February 1, 1987
Glucagon stimulates adenylate cyclase through GR2 glucagon receptors: a process which can be attenuated by glucagon stimulating inositol phospholipid metabolism through GR1 glucagon receptorsM D Houslay, M J Wakelam, G J Murphy, et al.
Journal of Clinical Pathology|August 30, 2005
Guillain Barré syndrome precipitated by the use of antilymphocyte globulin in the treatment of severe aplastic anaemiaB Kaya, C E Davies, H E Oakervee, et al.
The Biochemical Journal|April 1, 1989
Glucagon desensitization of adenylate cyclase and stimulation of inositol phospholipid metabolism does not involve the inhibitory guanine nucleotide regulatory protein Gi, which is inactivated upon challenge of hepatocytes with glucagonG J Murphy, D J Gawler, G Milligan, et al.
Pageof 5