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J H Robbins

Showing results (61-70 of 78) with videos related to

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Cancer Research|June 1, 1978
Ultraviolet light-induced sister chromatid exchanges in xeroderma pigmentosum and in Cockayne's syndrome lymphocyte cell linesW S Cheng, R E Tarone, A D Andrews, et al.
Journal of Neurology, Neurosurgery, and Psychiatry|September 1, 1985
Parkinson's disease and Alzheimer's disease: hypersensitivity to X rays in cultured cell linesJ H Robbins, F Otsuka, R E Tarone, et al.
Journal of Neuropathology and Experimental Neurology|September 1, 1985
Cockayne syndrome: clinicopathologic and tissue culture studies of affected siblingsR W Leech, R A Brumback, R H Miller, et al.
Mutation Research|January 1, 1986
Alzheimer disease fibroblasts are hypersensitive to the lethal effects of a DNA-damaging chemicalD A Scudiero, R J Polinsky, R A Brumback, et al.
The Journal of Biological Chemistry|March 5, 1993
Gene-specific DNA repair in xeroderma pigmentosum complementation groups A, C, D, and F. Relation to cellular survival and clinical featuresM K Evans, J H Robbins, M B Ganges, et al.
Photochemistry and Photobiology|May 1, 1983
Host cell reactivation by fibroblasts from patients with pigmentary degeneration of the retinaC D Lytle, R E Tarone, S F Barrett, et al.
Biochemical and Biophysical Research Communications|December 16, 1987
Alzheimer's disease fibroblasts have normal repair of N-methyl-N'-nitro-N-nitrosoguanidine-induced DNA damage determined by the alkaline elution techniqueT J Kinsella, P P Dobson, A J Fornace, et al.
Proceedings of the National Academy of Sciences of the United States of America|January 1, 1975
Genetic heterogeneity in xeroderma pigmentosum: complementation groups and their relationship to DNA repair ratesK H Kraemer, H G Coon, R A Petinga, et al.
Basic Life Sciences|January 1, 1985
Hypersensitivity to DNA-damaging agents in abiotrophies: a new explanation for degeneration of neurons, photoreceptors, and muscle in Alzheimer, Parkinson and Huntington diseases, retinitis pigmentosa, and Duchenne muscular dystrophyJ H Robbins, R A Brumback, R J Polinsky, et al.
Mutation Research|December 1, 1975
Five complementation groups in xeroderma pigmentosumK H Kraemer, E A De Weerd-Kastelein, J H Robbins, et al.
Pageof 8

Showing results (61-70 of 78) with videos related to

Sort By:
Pageof 8
Cancer Research|June 1, 1978
Ultraviolet light-induced sister chromatid exchanges in xeroderma pigmentosum and in Cockayne's syndrome lymphocyte cell linesW S Cheng, R E Tarone, A D Andrews, et al.
Journal of Neurology, Neurosurgery, and Psychiatry|September 1, 1985
Parkinson's disease and Alzheimer's disease: hypersensitivity to X rays in cultured cell linesJ H Robbins, F Otsuka, R E Tarone, et al.
Journal of Neuropathology and Experimental Neurology|September 1, 1985
Cockayne syndrome: clinicopathologic and tissue culture studies of affected siblingsR W Leech, R A Brumback, R H Miller, et al.
Mutation Research|January 1, 1986
Alzheimer disease fibroblasts are hypersensitive to the lethal effects of a DNA-damaging chemicalD A Scudiero, R J Polinsky, R A Brumback, et al.
The Journal of Biological Chemistry|March 5, 1993
Gene-specific DNA repair in xeroderma pigmentosum complementation groups A, C, D, and F. Relation to cellular survival and clinical featuresM K Evans, J H Robbins, M B Ganges, et al.
Photochemistry and Photobiology|May 1, 1983
Host cell reactivation by fibroblasts from patients with pigmentary degeneration of the retinaC D Lytle, R E Tarone, S F Barrett, et al.
Biochemical and Biophysical Research Communications|December 16, 1987
Alzheimer's disease fibroblasts have normal repair of N-methyl-N'-nitro-N-nitrosoguanidine-induced DNA damage determined by the alkaline elution techniqueT J Kinsella, P P Dobson, A J Fornace, et al.
Proceedings of the National Academy of Sciences of the United States of America|January 1, 1975
Genetic heterogeneity in xeroderma pigmentosum: complementation groups and their relationship to DNA repair ratesK H Kraemer, H G Coon, R A Petinga, et al.
Basic Life Sciences|January 1, 1985
Hypersensitivity to DNA-damaging agents in abiotrophies: a new explanation for degeneration of neurons, photoreceptors, and muscle in Alzheimer, Parkinson and Huntington diseases, retinitis pigmentosa, and Duchenne muscular dystrophyJ H Robbins, R A Brumback, R J Polinsky, et al.
Mutation Research|December 1, 1975
Five complementation groups in xeroderma pigmentosumK H Kraemer, E A De Weerd-Kastelein, J H Robbins, et al.
Pageof 8