Showing results (41-50 of 55) with videos related to

Sort By:
Pageof 6
The Journal of Biological Chemistry|March 30, 2001
Rantes activates Jak2 and Jak3 to regulate engagement of multiple signaling pathways in T cellsM Wong, S Uddin, B Majchrzak, et al.
Biochemical and Biophysical Research Communications|March 29, 2000
Interferon-dependent activation of the serine kinase PI 3'-kinase requires engagement of the IRS pathway but not the Stat pathwayS Uddin, B Majchrzak, P C Wang, et al.
Experimental Hematology|July 25, 2000
All-trans-retinoic acid induces tyrosine phosphorylation of the CrkL adapter in acute promyelocytic leukemia cellsY Alsayed, S Modi, S Uddin, et al.
Multiple Sclerosis (Houndmills, Basingstoke, England)|October 12, 2010
Interferon-beta is a key regulator of proinflammatory events in experimental autoimmune encephalomyelitisC L Galligan, L M Pennell, T T Murooka, et al.
Journal of Interferon Research|August 1, 1986
Antiviral and antitumor effects of a human interferon analog, IFN-alpha Con 1, assessed in hamstersB W Altrock, K D Fagin, H R Hockman, et al.
The Journal of Biological Chemistry|November 5, 1999
Catalytically active TYK2 is essential for interferon-beta-mediated phosphorylation of STAT3 and interferon-alpha receptor-1 (IFNAR-1) but not for activation of phosphoinositol 3-kinaseM R Rani, D W Leaman, Y Han, et al.
Biochemical and Biophysical Research Communications|June 9, 1997
Interaction of p59fyn with interferon-activated Jak kinasesS Uddin, D A Sher, Y Alsayed, et al.
Journal of Interferon & Cytokine Research : the Official Journal of the International Society for Interferon and Cytokine Research|August 24, 1999
Activation of the Jak-Stat pathway in cells that exhibit selective sensitivity to the antiviral effects of IFN-beta compared with IFN-alphaI M Grumbach, E N Fish, S Uddin, et al.
The Journal of Biological Chemistry|October 9, 1999
Activation of the p38 mitogen-activated protein kinase by type I interferonsS Uddin, B Majchrzak, J Woodson, et al.
Oncogene|July 4, 2017
Human SLFN5 is a transcriptional co-repressor of STAT1-mediated interferon responses and promotes the malignant phenotype in glioblastomaA D Arslan, A Sassano, D Saleiro, et al.
Pageof 6