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Hypertension (Dallas, Tex. : 1979)|October 1, 1993
Targeting gene expression to specific cardiovascular cell types in transgenic miceJ J Hunter, H Zhu, K J Lee, et al.The Journal of Biological Chemistry|September 29, 1995
Ventricular expression of a MLC-2v-ras fusion gene induces cardiac hypertrophy and selective diastolic dysfunction in transgenic miceJ J Hunter, N Tanaka, H A Rockman, et al.The Journal of Biological Chemistry|May 30, 1997
The MEKK-JNK pathway is stimulated by alpha1-adrenergic receptor and ras activation and is associated with in vitro and in vivo cardiac hypertrophyM T Ramirez, V P Sah, X L Zhao, et al.Proceedings of the National Academy of Sciences of the United States of America|April 29, 1997
Ras-dependent pathways induce obstructive hypertrophy in echo-selected transgenic miceK R Gottshall, J J Hunter, N Tanaka, et al.Journal of Molecular and Cellular Cardiology|January 30, 1999
Distinct molecular phenotypes in murine cardiac muscle development, growth, and hypertrophyJ R Schoenfeld, M Vasser, P Jhurani, et al.Cell|February 7, 1997
MLP-deficient mice exhibit a disruption of cardiac cytoarchitectural organization, dilated cardiomyopathy, and heart failureS Arber, J J Hunter, J Ross, et al.American Journal of Physiology. Heart and Circulatory Physiology|May 18, 2001
Effects of deletion of muscle LIM protein on myocyte functionZ Su, A Yao, I Zubair, et al.The American Journal of Physiology|September 1, 1995
Cardiac muscle diseases in genetically engineered mice: evolution of molecular physiologyK R ChienSeminars in Oncology|March 10, 2001
Myocyte survival pathways and cardiomyopathy: implications for trastuzumab cardiotoxicityK R ChienPageof 16