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J J Lebrun

Showing results (1-10 of 23) with videos related to

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Molecular and Cellular Biology|March 1, 1997
Activin and inhibin have antagonistic effects on ligand-dependent heteromerization of the type I and type II activin receptors and human erythroid differentiationJ J Lebrun, W W Vale
Proceedings of the National Academy of Sciences of the United States of America|November 12, 1996
Regulation of transforming growth factor beta- and activin-induced transcription by mammalian Mad proteinsY Chen, J J Lebrun, W Vale
The Journal of Biological Chemistry|March 30, 2001
The p38 MAPK pathway is required for cell growth inhibition of human breast cancer cells in response to activinE Cocolakis, S Lemay, S Ali, et al.
Molecular Endocrinology (Baltimore, Md.)|January 19, 1999
Roles of pathway-specific and inhibitory Smads in activin receptor signalingJ J Lebrun, K Takabe, Y Chen, et al.
Cell Death & Disease|October 13, 2012
A transcriptionally active pRb-E2F1-P/CAF signaling pathway is central to TGFβ-mediated apoptosisJ Korah, N Falah, A Lacerte, et al.
The Journal of Biological Chemistry|May 5, 1995
Proline-rich sequence-mediated Jak2 association to the prolactin receptor is required but not sufficient for signal transductionJ J Lebrun, S Ali, A Ullrich, et al.
Molecular Endocrinology (Baltimore, Md.)|July 1, 1992
Expression of prolactin and its receptor in human lymphoid cellsI Pellegrini, J J Lebrun, S Ali, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 29, 2001
Inactivation of menin, a Smad3-interacting protein, blocks transforming growth factor type beta signalingH Kaji, L Canaff, J J Lebrun, et al.
Proceedings of the National Academy of Sciences of the United States of America|April 25, 1995
A single phosphotyrosine residue of the prolactin receptor is responsible for activation of gene transcriptionJ J Lebrun, S Ali, V Goffin, et al.
The Journal of Biological Chemistry|May 13, 1994
Prolactin-induced proliferation of Nb2 cells involves tyrosine phosphorylation of the prolactin receptor and its associated tyrosine kinase JAK2J J Lebrun, S Ali, L Sofer, et al.
Pageof 3

Showing results (1-10 of 23) with videos related to

Sort By:
Pageof 3
Molecular and Cellular Biology|March 1, 1997
Activin and inhibin have antagonistic effects on ligand-dependent heteromerization of the type I and type II activin receptors and human erythroid differentiationJ J Lebrun, W W Vale
Proceedings of the National Academy of Sciences of the United States of America|November 12, 1996
Regulation of transforming growth factor beta- and activin-induced transcription by mammalian Mad proteinsY Chen, J J Lebrun, W Vale
The Journal of Biological Chemistry|March 30, 2001
The p38 MAPK pathway is required for cell growth inhibition of human breast cancer cells in response to activinE Cocolakis, S Lemay, S Ali, et al.
Molecular Endocrinology (Baltimore, Md.)|January 19, 1999
Roles of pathway-specific and inhibitory Smads in activin receptor signalingJ J Lebrun, K Takabe, Y Chen, et al.
Cell Death & Disease|October 13, 2012
A transcriptionally active pRb-E2F1-P/CAF signaling pathway is central to TGFβ-mediated apoptosisJ Korah, N Falah, A Lacerte, et al.
The Journal of Biological Chemistry|May 5, 1995
Proline-rich sequence-mediated Jak2 association to the prolactin receptor is required but not sufficient for signal transductionJ J Lebrun, S Ali, A Ullrich, et al.
Molecular Endocrinology (Baltimore, Md.)|July 1, 1992
Expression of prolactin and its receptor in human lymphoid cellsI Pellegrini, J J Lebrun, S Ali, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 29, 2001
Inactivation of menin, a Smad3-interacting protein, blocks transforming growth factor type beta signalingH Kaji, L Canaff, J J Lebrun, et al.
Proceedings of the National Academy of Sciences of the United States of America|April 25, 1995
A single phosphotyrosine residue of the prolactin receptor is responsible for activation of gene transcriptionJ J Lebrun, S Ali, V Goffin, et al.
The Journal of Biological Chemistry|May 13, 1994
Prolactin-induced proliferation of Nb2 cells involves tyrosine phosphorylation of the prolactin receptor and its associated tyrosine kinase JAK2J J Lebrun, S Ali, L Sofer, et al.
Pageof 3