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Shock (Augusta, Ga.)|June 5, 2020
Indirect, Non-Thermal Atmospheric Plasma Promotes Bacterial Killing in vitro and Wound Disinfection in vivo Using Monogenic and Polygenic Models of Type 2 Diabetes (Without Adverse Metabolic Complications)Chris R Cooley, John Mark McLain, Samuel D Dupuy, et al.
Plos One|October 12, 2012
Regulation of the CCL2 gene in pancreatic β-cells by IL-1β and glucocorticoids: role of MKP-1Susan J Burke, Matthew R Goff, Barrett L Updegraff, et al.
Chemical Science|June 25, 2021
α-CGRP disrupts amylin fibrillization and regulates insulin secretion: implications on diabetes and migraineAmber L H Gray, Aleksandra Antevska, Benjamin A Link, et al.
Biochimica Et Biophysica Acta|April 18, 2015
CCL20 is elevated during obesity and differentially regulated by NF-κB subunits in pancreatic β-cellsSusan J Burke, Michael D Karlstad, Kellie M Regal, et al.
Molecules (Basel, Switzerland)|April 13, 2024
Channel Expansion in the Ligand-Binding Domain of the Glucocorticoid Receptor Contributes to the Activity of Highly Potent Glucocorticoid AnaloguesWesley B Seaton, Susan J Burke, Alexander R Fisch, et al.
The Journal of Biological Chemistry|April 9, 2015
Thiobenzothiazole-modified Hydrocortisones Display Anti-inflammatory Activity with Reduced Impact on Islet β-Cell FunctionSusan J Burke, Amanda L May, Robert C Noland, et al.
Journal of Applied Physiology (Bethesda, Md. : 1985)|May 17, 2019
Low-intensity exercise induces acute shifts in liver and skeletal muscle substrate metabolism but not chronic adaptations in tissue oxidative capacityScott E Fuller, Tai-Yu Huang, Jacob Simon, et al.
Molecular Metabolism|June 9, 2023
Pharmacological inhibition of lipolysis prevents adverse metabolic outcomes during glucocorticoid administrationMelissa A Linden, Susan J Burke, Humza A Pirzadah, et al.
Diabetes|May 24, 2023
GDF15 Mediates the Effect of Skeletal Muscle Contraction on Glucose-Stimulated Insulin SecretionHui Zhang, Anny Mulya, Stephan Nieuwoudt, et al.
ACS Medicinal Chemistry Letters|October 22, 2021
Potent Anti-Inflammatory, Arylpyrazole-Based Glucocorticoid Receptor Agonists That Do Not Impair Insulin SecretionBrandon J Kennedy, Ashley M Lato, Alexander R Fisch, et al.
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