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Nature Communications|March 1, 2019
Tissue-resident Eomes+ NK cells are the major innate lymphoid cell population in human infant intestineAdrian F Sagebiel, Fenja Steinert, Sebastian Lunemann, et al.Journal of Immunology (Baltimore, Md. : 1950)|March 21, 2003
Viremia control despite escape from a rapid and potent autologous neutralizing antibody response after therapy cessation in an HIV-1-infected individualDavid C Montefiori, Marcus Altfeld, Paul K Lee, et al.Plos One|October 4, 2011
Changes in cytokine levels and NK cell activation associated with influenzaStephanie Jost, Heloise Quillay, Jeff Reardon, et al.Plos Pathogens|June 24, 2022
HIV-1 Nef-mediated downregulation of CD155 results in viral restriction by KIR2DL5+ NK cellsPia Fittje, Angelique Hœlzemer, Wilfredo F Garcia-Beltran, et al.Journal of Translational Medicine|May 25, 2004
CD8+ T lymphocyte responses target functionally important regions of Protease and Integrase in HIV-1 infected subjectsWilliam R Rodriguez, Marylyn M Addo, Almas Rathod, et al.Retrovirology|July 23, 2013
Dysregulated Tim-3 expression on natural killer cells is associated with increased Galectin-9 levels in HIV-1 infectionStephanie Jost, Uriel Y Moreno-Nieves, Wilfredo F Garcia-Beltran, et al.Current HIV Research|May 14, 2008
Relative dominance of Env-gp41-specific cytotoxic T lymphocytes responses in HIV-1 advanced infectionYan Zhuang, Yongtao Sun, Song Zhai, et al.AIDS (London, England)|December 4, 2020
HIV-1 evades a Gag mutation that abrogates killer cell immunoglobulin-like receptor binding and disinhibits natural killer cells in infected individuals with KIR2DL2+/HLA-C*03: 04+ genotypeMaja C Ziegler, Kewreshini Naidoo, Anais Chapel, et al.Frontiers in Immunology|December 18, 2020
Distinct Signatures in the Receptor Repertoire Discriminate CD56bright and CD56dim Natural Killer CellsVera Schwane, Van Hung Huynh-Tran, Sarah Vollmers, et al.European Journal of Immunology|July 22, 2014
Increased frequency and function of KIR2DL1-3⁺ NK cells in primary HIV-1 infection are determined by HLA-C group haplotypesChristian Körner, Mitchell E Granoff, Molly A Amero, et al.Pageof 29