Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

J K Parks

Showing results (21-30 of 46) with videos related to

Pageof 5
Sort By:
Biochimica Et Biophysica Acta|December 14, 1999
Mitochondrial impact on nerve growth factor production in vascular smooth muscle-derived cellsT B Sherer, P S Neff, J K Parks, et al.
Biochimica Et Biophysica Acta|February 16, 1999
The parkinsonian neurotoxin MPP+ opens the mitochondrial permeability transition pore and releases cytochrome c in isolated mitochondria via an oxidative mechanismD S Cassarino, J K Parks, W D Parker, et al.
Neurology|January 1, 1996
Association of the mitochondrial 8344 MERRF mutation with maternally inherited spinocerebellar degeneration and Leigh diseaseN Howell, I Kubacka, R Smith, et al.
Biochemical Medicine|April 1, 1979
Loosely coupled mitochondrial oxidative phosphorylation induced by protoporphyrinD A Stumpf, E R McCabe, J K Parks, et al.
Biochemical Medicine|April 1, 1982
A mechanism of toxicity of isovaleric acid in rat liver mitochondriaB J Bergen, D A Stumpf, R Haas, et al.
Muscle & Nerve|January 1, 1982
Protonmotive force in muscle mitochondriaD A Stumpf, R Haas, L A Eguren, et al.
Journal of Geriatric Psychiatry and Neurology|April 11, 1992
Cytochrome oxidase inhibition: a novel animal model of Alzheimer's diseaseM C Bennett, D M Diamond, S L Stryker, et al.
Journal of Neurochemistry|February 22, 2001
Neurotoxic Abeta peptides increase oxidative stress in vivo through NMDA-receptor and nitric-oxide-synthase mechanisms, and inhibit complex IV activity and induce a mitochondrial permeability transition in vitroJ K Parks, T S Smith, P A Trimmer, et al.
Biochemical and Biophysical Research Communications|July 24, 1998
Cyclosporin A increases resting mitochondrial membrane potential in SY5Y cells and reverses the depressed mitochondrial membrane potential of Alzheimer's disease cybridsD S Cassarino, R H Swerdlow, J K Parks, et al.
The Journal of Pediatrics|January 1, 1984
Lipoamide dehydrogenase deficiency with primary lactic acidosis: favorable response to treatment with oral lipoic acidR Matalon, D A Stumpf, K Michals, et al.
Pageof 5

Showing results (21-30 of 46) with videos related to

Sort By:
Pageof 5
Biochimica Et Biophysica Acta|December 14, 1999
Mitochondrial impact on nerve growth factor production in vascular smooth muscle-derived cellsT B Sherer, P S Neff, J K Parks, et al.
Biochimica Et Biophysica Acta|February 16, 1999
The parkinsonian neurotoxin MPP+ opens the mitochondrial permeability transition pore and releases cytochrome c in isolated mitochondria via an oxidative mechanismD S Cassarino, J K Parks, W D Parker, et al.
Neurology|January 1, 1996
Association of the mitochondrial 8344 MERRF mutation with maternally inherited spinocerebellar degeneration and Leigh diseaseN Howell, I Kubacka, R Smith, et al.
Biochemical Medicine|April 1, 1979
Loosely coupled mitochondrial oxidative phosphorylation induced by protoporphyrinD A Stumpf, E R McCabe, J K Parks, et al.
Biochemical Medicine|April 1, 1982
A mechanism of toxicity of isovaleric acid in rat liver mitochondriaB J Bergen, D A Stumpf, R Haas, et al.
Muscle & Nerve|January 1, 1982
Protonmotive force in muscle mitochondriaD A Stumpf, R Haas, L A Eguren, et al.
Journal of Geriatric Psychiatry and Neurology|April 11, 1992
Cytochrome oxidase inhibition: a novel animal model of Alzheimer's diseaseM C Bennett, D M Diamond, S L Stryker, et al.
Journal of Neurochemistry|February 22, 2001
Neurotoxic Abeta peptides increase oxidative stress in vivo through NMDA-receptor and nitric-oxide-synthase mechanisms, and inhibit complex IV activity and induce a mitochondrial permeability transition in vitroJ K Parks, T S Smith, P A Trimmer, et al.
Biochemical and Biophysical Research Communications|July 24, 1998
Cyclosporin A increases resting mitochondrial membrane potential in SY5Y cells and reverses the depressed mitochondrial membrane potential of Alzheimer's disease cybridsD S Cassarino, R H Swerdlow, J K Parks, et al.
The Journal of Pediatrics|January 1, 1984
Lipoamide dehydrogenase deficiency with primary lactic acidosis: favorable response to treatment with oral lipoic acidR Matalon, D A Stumpf, K Michals, et al.
Pageof 5