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J Kaiser

Showing results (521-530 of 621) with videos related to

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Plos Genetics|December 21, 2017
Regulation of the cohesin-loading factor NIPBL: Role of the lncRNA NIPBL-AS1 and identification of a distal enhancer elementJessica Zuin, Valentina Casa, Jelena Pozojevic, et al.
The International Journal of Cardiovascular Imaging|November 26, 2022
Subclinical myocardial dysfunction is revealed by speckle tracking echocardiography in patients with Cornelia de Lange syndromeLaura Trujillano, Ariadna Ayerza-Casas, Beatriz Puisac, et al.
International Journal of Molecular Sciences|January 8, 2023
G Protein-Coupled Receptor 15 Expression Is Associated with Myocardial InfarctionTina Haase, Christian Müller, Bastian Stoffers, et al.
Journal of Immunology (Baltimore, Md. : 1950)|November 13, 2012
Cutting edge: FAS (CD95) mediates noncanonical IL-1β and IL-18 maturation via caspase-8 in an RIP3-independent mannerLukas Bossaller, Ping-I Chiang, Christian Schmidt-Lauber, et al.
Plos Genetics|March 29, 2014
Coronary heart disease-associated variation in TCF21 disrupts a miR-224 binding site and miRNA-mediated regulationClint L Miller, Ulrike Haas, Roxanne Diaz, et al.
The Journal of Pathology|October 2, 2021
Homozygous WASHC4 variant in two sisters causes a syndromic phenotype defined by dysmorphisms, intellectual disability, profound developmental disorder, and skeletal muscle involvementAndrea Gangfuß, Artur Czech, Andreas Hentschel, et al.
Journal of Immunology (Baltimore, Md. : 1950)|July 27, 2014
Caspase-8 modulates dectin-1 and complement receptor 3-driven IL-1β production in response to β-glucans and the fungal pathogen, Candida albicansSandhya Ganesan, Vijay A K Rathinam, Lukas Bossaller, et al.
Journal of Immunology (Baltimore, Md. : 1950)|May 14, 2014
Cutting Edge: RIP1 kinase activity is dispensable for normal development but is a key regulator of inflammation in SHARPIN-deficient miceScott B Berger, Viera Kasparcova, Sandy Hoffman, et al.
Proceedings of the National Academy of Sciences of the United States of America|May 14, 2014
RIP1 suppresses innate immune necrotic as well as apoptotic cell death during mammalian parturitionWilliam J Kaiser, Lisa P Daley-Bauer, Roshan J Thapa, et al.
Iscience|August 9, 2023
Down syndrome is associated with altered frequency and functioning of tracheal multiciliated cells, and response to influenza virus infectionSamantha N Thomas, Brian F Niemeyer, Rocio J Jimenez-Valdes, et al.
Pageof 63

Showing results (521-530 of 621) with videos related to

Sort By:
Pageof 63
Plos Genetics|December 21, 2017
Regulation of the cohesin-loading factor NIPBL: Role of the lncRNA NIPBL-AS1 and identification of a distal enhancer elementJessica Zuin, Valentina Casa, Jelena Pozojevic, et al.
The International Journal of Cardiovascular Imaging|November 26, 2022
Subclinical myocardial dysfunction is revealed by speckle tracking echocardiography in patients with Cornelia de Lange syndromeLaura Trujillano, Ariadna Ayerza-Casas, Beatriz Puisac, et al.
International Journal of Molecular Sciences|January 8, 2023
G Protein-Coupled Receptor 15 Expression Is Associated with Myocardial InfarctionTina Haase, Christian Müller, Bastian Stoffers, et al.
Journal of Immunology (Baltimore, Md. : 1950)|November 13, 2012
Cutting edge: FAS (CD95) mediates noncanonical IL-1β and IL-18 maturation via caspase-8 in an RIP3-independent mannerLukas Bossaller, Ping-I Chiang, Christian Schmidt-Lauber, et al.
Plos Genetics|March 29, 2014
Coronary heart disease-associated variation in TCF21 disrupts a miR-224 binding site and miRNA-mediated regulationClint L Miller, Ulrike Haas, Roxanne Diaz, et al.
The Journal of Pathology|October 2, 2021
Homozygous WASHC4 variant in two sisters causes a syndromic phenotype defined by dysmorphisms, intellectual disability, profound developmental disorder, and skeletal muscle involvementAndrea Gangfuß, Artur Czech, Andreas Hentschel, et al.
Journal of Immunology (Baltimore, Md. : 1950)|July 27, 2014
Caspase-8 modulates dectin-1 and complement receptor 3-driven IL-1β production in response to β-glucans and the fungal pathogen, Candida albicansSandhya Ganesan, Vijay A K Rathinam, Lukas Bossaller, et al.
Journal of Immunology (Baltimore, Md. : 1950)|May 14, 2014
Cutting Edge: RIP1 kinase activity is dispensable for normal development but is a key regulator of inflammation in SHARPIN-deficient miceScott B Berger, Viera Kasparcova, Sandy Hoffman, et al.
Proceedings of the National Academy of Sciences of the United States of America|May 14, 2014
RIP1 suppresses innate immune necrotic as well as apoptotic cell death during mammalian parturitionWilliam J Kaiser, Lisa P Daley-Bauer, Roshan J Thapa, et al.
Iscience|August 9, 2023
Down syndrome is associated with altered frequency and functioning of tracheal multiciliated cells, and response to influenza virus infectionSamantha N Thomas, Brian F Niemeyer, Rocio J Jimenez-Valdes, et al.
Pageof 63