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Arteriosclerosis, Thrombosis, and Vascular Biology|April 1, 1997
Phenotypic variation in patients heterozygous for familial defective apolipoprotein B (FDB) in three European countriesP S Hansen, J C Defesche, J J Kastelein, et al.
AIDS (London, England)|December 12, 2001
Nevirapine-containing antiretroviral therapy in HIV-1 infected patients results in an anti-atherogenic lipid profileM van der Valk, J J Kastelein, R L Murphy, et al.
European Journal of Preventive Cardiology|May 27, 2011
Asymmetrical distribution of atherosclerosis in the carotid artery: identical patterns across age, race, and genderParvin Tajik, Rudy Meijer, Raphaël Duivenvoorden, et al.
Journal of Lipid Research|April 7, 1999
Lipoprotein lipase activity is decreased in a large cohort of patients with coronary artery disease and is associated with changes in lipids and lipoproteinsH E Henderson, J J Kastelein, A H Zwinderman, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|July 9, 2005
Complete rescue of lipoprotein lipase-deficient mice by somatic gene transfer of the naturally occurring LPLS447X beneficial mutationColin J D Ross, Guoqing Liu, Jan Albert Kuivenhoven, et al.
Clinical Genetics|January 15, 2000
Two common mutations (D9N, N291S) in lipoprotein lipase: a cumulative analysis of their influence on plasma lipids and lipoproteins in men and womenJ J Kastelein, J M Ordovas, M E Wittekoek, et al.
Blood|June 10, 2009
AAV-1-mediated gene transfer to skeletal muscle in humans results in dose-dependent activation of capsid-specific T cellsFederico Mingozzi, Janneke J Meulenberg, Daniel J Hui, et al.
Nature Genetics|September 10, 1998
Assignment of Tangier disease to chromosome 9q31 by a graphical linkage exclusion strategyS Rust, M Walter, H Funke, et al.
Diabetes & Vascular Disease Research|March 18, 2015
Design and baseline characteristics of the CARdiovascular Outcome Trial of LINAgliptin Versus Glimepiride in Type 2 Diabetes (CAROLINA®)Nikolaus Marx, Julio Rosenstock, Steven E Kahn, et al.
Circulation|June 17, 1997
Genetic variant showing a positive interaction with beta-blocking agents with a beneficial influence on lipoprotein lipase activity, HDL cholesterol, and triglyceride levels in coronary artery disease patients. The Ser447-stop substitution in the lipoprotein lipase gene. REGRESS Study GroupB E Groenemeijer, M D Hallman, P W Reymer, et al.
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