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Molecular Cell|July 14, 1998
Human Smad3 and Smad4 are sequence-specific transcription activatorsL Zawel, J L Dai, P Buckhaults, et al.
Molecular Carcinogenesis|September 16, 1999
Transforming growth factor-beta responsiveness in DPC4/SMAD4-null cancer cellsJ L Dai, M Schutte, R K Bansal, et al.
International Journal of Antimicrobial Agents|June 28, 2008
Prediction of antibiotic resistance proteins from sequence-derived properties irrespective of sequence similarityH L Zhang, H H Lin, L Tao, et al.
Proceedings of the National Academy of Sciences of the United States of America|April 16, 1998
Targeted deletion of Smad4 shows it is required for transforming growth factor beta and activin signaling in colorectal cancer cellsS Zhou, P Buckhaults, L Zawel, et al.
Genetics and Molecular Research : GMR|May 14, 2016
Agrobacterium-mediated transformation of the β-subunit gene in 7S globulin protein in soybean using RNAi technologyJ Qu, S Y Liu, P W Wang, et al.
The American Journal of Pathology|January 7, 2000
Immunohistochemical labeling for dpc4 mirrors genetic status in pancreatic adenocarcinomas : a new marker of DPC4 inactivationR E Wilentz, G H Su, J L Dai, et al.
The Prostate|March 8, 2008
RANKL inhibition is an effective adjuvant for docetaxel in a prostate cancer bone metastases modelK M Woods Ignatoski, J F Escara-Wilke, J-L Dai, et al.
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