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Stroke|April 1, 1992
A rat model of severe neonatal hypoxic-ischemic brain injuryP H Schwartz, W F Massarweh, H V Vinters, et al.Stroke|April 1, 1992
Protective effects of felbamate against hypoxia in the rat hippocampal sliceR A Wallis, K L Panizzon, M D Fairchild, et al.The American Journal of Physiology|June 1, 1989
Local cerebral glucose utilization during status epilepticus in newborn primatesD G Fujikawa, B E Dwyer, R R Lake, et al.Brain Research|June 28, 1988
Generalized seizures deplete brain energy reserves in normoxemic newborn monkeysD G Fujikawa, R C Vannucci, B E Dwyer, et al.Journal of Neurochemistry|July 1, 1988
Low [3H]cytochalasin B binding in the cerebral cortex of newborn ratA M Morin, B E Dwyer, D G Fujikawa, et al.Brain Research|November 6, 1992
Low sodium injury in the hippocampal slice is mediated through NMDA receptorsJ E Parsons, R A Wallis, K L Panizzon, et al.Brain Research|August 14, 1999
Self-sustaining status epilepticus after a brief electrical stimulation of the perforant path: a 2-deoxyglucose studyA Pereira de Vasconcelos, A M Mazarati, C G Wasterlain, et al.Proceedings of the National Academy of Sciences of the United States of America|April 29, 1999
Substance P is expressed in hippocampal principal neurons during status epilepticus and plays a critical role in the maintenance of status epilepticusH Liu, A M Mazarati, H Katsumori, et al.Experimental Eye Research|September 1, 1988
Localization of retinal calmodulin kinaseJ M Bronstein, C G Wasterlain, D Bok, et al.Neuroscience|November 14, 2000
Patterns of status epilepticus-induced substance P expression during developmentH Liu, R Sankar, D H Shin, et al.Pageof 41