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J L Moake

Showing results (71-80 of 123) with videos related to

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Transfusion|May 1, 1978
Hemolysis induced by cefazolin and cephalothin in a patient with penicillin sensitivityJ L Moake, C F Butler, G M Hewell, et al.
Blood|May 1, 1988
Shear-induced platelet aggregation can be mediated by vWF released from platelets, as well as by exogenous large or unusually large vWF multimers, requires adenosine diphosphate, and is resistant to aspirinJ L Moake, N A Turner, N A Stathopoulos, et al.
The American Journal of Physiology|June 1, 1989
Cryosupernatant regulates accumulation of unusually large vWF multimers from endothelial cellsJ A Frangos, J L Moake, L Nolasco, et al.
Lancet (London, England)|July 23, 1988
Lytic anti-endothelial cell antibodies in haemolytic-uraemic syndromeD Y Leung, J L Moake, P L Havens, et al.
Blood|May 1, 1980
Prothrombin Houston: a dysprothrombin identifiable by crossed immunoelectrofocusing and abnormal Echis carinatus venom activationR S Weinger, C Rudy, J L Moake, et al.
American Journal of Hematology|January 1, 1980
Fibrinogen Houston: a dysfibrinogen exhibiting defective fibrin monomer aggregation and alpha-chain cross-linkagesR S Weinger, C Rudy, J L Moake, et al.
The Journal of Clinical Investigation|November 1, 1991
von Willebrand factor binding to platelet GpIb initiates signals for platelet activationM H Kroll, T S Harris, J L Moake, et al.
Transactions - American Society for Artificial Internal Organs|January 1, 1980
Effects of antiplatelet agents on platelets exposed to shear stressR A Hardwick, J D Hellums, J L Moake, et al.
Blood|September 1, 1996
Platelets and shear stressM H Kroll, J D Hellums, L V McIntire, et al.
The Journal of Pediatrics|January 1, 1992
Chronic relapsing thrombotic thrombocytopenic purpura in infants with large von Willebrand factor multimers during remissionM M Chintagumpala, R L Hurwitz, J L Moake, et al.
Pageof 13

Showing results (71-80 of 123) with videos related to

Sort By:
Pageof 13
Transfusion|May 1, 1978
Hemolysis induced by cefazolin and cephalothin in a patient with penicillin sensitivityJ L Moake, C F Butler, G M Hewell, et al.
Blood|May 1, 1988
Shear-induced platelet aggregation can be mediated by vWF released from platelets, as well as by exogenous large or unusually large vWF multimers, requires adenosine diphosphate, and is resistant to aspirinJ L Moake, N A Turner, N A Stathopoulos, et al.
The American Journal of Physiology|June 1, 1989
Cryosupernatant regulates accumulation of unusually large vWF multimers from endothelial cellsJ A Frangos, J L Moake, L Nolasco, et al.
Lancet (London, England)|July 23, 1988
Lytic anti-endothelial cell antibodies in haemolytic-uraemic syndromeD Y Leung, J L Moake, P L Havens, et al.
Blood|May 1, 1980
Prothrombin Houston: a dysprothrombin identifiable by crossed immunoelectrofocusing and abnormal Echis carinatus venom activationR S Weinger, C Rudy, J L Moake, et al.
American Journal of Hematology|January 1, 1980
Fibrinogen Houston: a dysfibrinogen exhibiting defective fibrin monomer aggregation and alpha-chain cross-linkagesR S Weinger, C Rudy, J L Moake, et al.
The Journal of Clinical Investigation|November 1, 1991
von Willebrand factor binding to platelet GpIb initiates signals for platelet activationM H Kroll, T S Harris, J L Moake, et al.
Transactions - American Society for Artificial Internal Organs|January 1, 1980
Effects of antiplatelet agents on platelets exposed to shear stressR A Hardwick, J D Hellums, J L Moake, et al.
Blood|September 1, 1996
Platelets and shear stressM H Kroll, J D Hellums, L V McIntire, et al.
The Journal of Pediatrics|January 1, 1992
Chronic relapsing thrombotic thrombocytopenic purpura in infants with large von Willebrand factor multimers during remissionM M Chintagumpala, R L Hurwitz, J L Moake, et al.
Pageof 13