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The Journal of Biological Chemistry|August 25, 1987
Activation of alpha 1-adrenoceptors, protein kinase C, or treatment with intracellular free Ca2+ elevating agents increases pineal phospholipase A2 activity. Evidence that protein kinase C may participate in Ca2+-dependent alpha 1-adrenergic stimulation of pineal phospholipase A2 activityA K Ho, D C KleinJournal of Neurochemistry|April 1, 1987
Phosphatidylinositol phosphodiesterase (phospholipase C) activity in the pineal gland: characterization and photoneural regulationA K Ho, D C KleinEndocrinology|September 1, 1976
Sympathetic nerve endings in the pineal gland protect against acute stress-induced increase in N-acetyltransferase (EC 2.3.1.5.) activityA G Parfitt, D C KleinScience (New York, N.Y.)|January 23, 1976
Melatonin inhibition of the neonatal pituitary response to luteinizing hormone-releasing factorJ E Martin, D C KleinEndocrinology|November 1, 1976
Long-term organ culture of rat anterior pituitary glandsJ E Martin, D C KleinBrain Research|April 7, 1980
Taurine release from the pineal-gland is stimulated via a beta-adrenergic mechanismG H Wheler, D C KleinEndocrinology|February 1, 1978
Transport of maternal[3H]melatonin to suckling rats and the fate of [3H]melatonin in the neonatal ratS M Reppert, D C KleinJournal of Neurochemistry|February 1, 1995
Stimulation of cyclic GMP accumulation by sodium nitroprusside is potentiated via a Gs mechanism in intact pinealocytesB H White, D C KleinMolecular Pharmacology|March 1, 1995
Norepinephrine stimulation of pineal cyclic AMP response element-binding protein phosphorylation: primary role of a beta-adrenergic receptor/cyclic AMP mechanismP H Roseboom, D C KleinPageof 20