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European Cytokine Network|November 1, 1994
Interleukin-1, tumor necrosis factor and their specific inhibitorsJ M Dayer, D BurgerThe Journal of Biological Chemistry|July 21, 1998
Direct contact between T lymphocytes and human dermal fibroblasts or synoviocytes down-regulates types I and III collagen production via cell-associated cytokinesR Rezzonico, D Burger, J M DayerEuropean Cytokine Network|January 16, 1999
Exposure of T lymphocytes to leflunomide but not to dexamethasone favors the production by monocytic cells of interleukin-1 receptor antagonist and the tissue-inhibitor of metalloproteinases-1 over that of interleukin-1beta and metalloproteinasesV Déage, D Burger, J M DayerEuropean Journal of Immunology|October 1, 1996
Expression and cleavage of tumor necrosis factor-alpha and tumor necrosis factor receptors by human monocytic cell lines upon direct contact with stimulated T cellsE Vey, D Burger, J M DayerEuropean Cytokine Network|February 12, 1998
Interferon-beta not only inhibits interleukin-1beta and tumor necrosis factor-alpha but stimulates interleukin-1 receptor antagonist production in human peripheral blood mononuclear cellsJ Coclet-Ninin, J M Dayer, D BurgerEuropean Journal of Immunology|January 1, 1997
Human Th1 cells preferentially induce interleukin (IL)-1beta while Th2 cells induce IL-1 receptor antagonist production upon cell/cell contact with monocytesC Chizzolini, R Chicheportiche, D Burger, et al.Current Opinion in Rheumatology|May 3, 2001
Anti-interleukin-1 therapy in rheumatic diseasesJ M Dayer, U Feige, C K Edwards, et al.The Journal of Clinical Investigation|July 1, 1995
The inhibitory activity of human interleukin-1 receptor antagonist is enhanced by type II interleukin-1 soluble receptor and hindered by type I interleukin-1 soluble receptorD Burger, R Chicheportiche, J G Giri, et al.Pageof 48