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American Journal of Physiology. Endocrinology and Metabolism|October 23, 2008
Fibroblast growth factor receptor 1 is a key regulator of early adipogenic events in human preadipocytesC H Widberg, F S Newell, A W Bachmann, et al.Australian and New Zealand Journal of Medicine|February 1, 1989
The effects of long term gliclazide administration on insulin secretion and insulin sensitivityA Ma, M Kamp, D Bird, et al.Biochemical and Biophysical Research Communications|June 26, 1995
G protein mutations in tumours of the pituitary, parathyroid and endocrine pancreasC V Boothroyd, S M Grimmond, D P Cameron, et al.The Journal of Pathology|February 1, 1996
Beta-cell apoptosis is responsible for the development of IDDM in the multiple low-dose streptozotocin modelB A O'Brien, B V Harmon, D P Cameron, et al.Biochemical and Biophysical Research Communications|November 30, 1994
Human adipocyte apoptosis occurs in malignancyJ B Prins, N I Walker, C M Winterford, et al.Biochemical and Biophysical Research Communications|June 15, 1994
Apoptosis of human adipocytes in vitroJ B Prins, N I Walker, C M Winterford, et al.The Journal of Clinical Endocrinology and Metabolism|November 1, 1976
A bioassay for NSILA-S in individual serum samples and its relationship to somatotropinR C Franklin, G C Rennie, H G Burger, et al.Diabetes|May 1, 1997
Apoptosis is the mode of beta-cell death responsible for the development of IDDM in the nonobese diabetic (NOD) mouseB A O'Brien, B V Harmon, D P Cameron, et al.The Journal of Clinical Endocrinology and Metabolism|April 1, 1979
Acid-ethanol-extractable nonsuppressible insulin-like activity (NSILA-S) during pregnancy and the puererium, and in cord serum at termR C Franklin, R J Pepperell, G C Rennie, et al.The Journal of Pathology|April 18, 2000
Nicotinamide prevents the development of diabetes in the cyclophosphamide-induced NOD mouse model by reducing beta-cell apoptosisB A O'Brien, B V Harmon, D P Cameron, et al.Pageof 6