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Neurological Research|November 5, 1997
beta-Amyloid induces cerebrovascular endothelial dysfunction in the rat brainJ M Price, E T Sutton, A Hellermann, et al.Journal of Cardiovascular Pharmacology|October 23, 1997
Beta-amyloid-induced coronary artery vasoactivity and endothelial damageT Thomas, E T Sutton, A Hellermann, et al.Life Sciences|January 1, 1997
Inhibition of cGMP mediated relaxation in small rat coronary arteries by block of CA++ activated K+ channelsJ M Price, A HellermannNeurological Research|July 16, 1999
The protective effect of K+ channel openers on beta-amyloid induced cerebrovascular endothelial dysfunctionX Chi, E T Sutton, T Thomas, et al.Life Sciences|January 1, 1996
Inhibition of cAMP mediated relaxation in rat coronary vessels by block of Ca++ activated K+ channelsJ M Price, J F Cabell, A HellermannJournal of Submicroscopic Cytology and Pathology|February 24, 2000
Estrogen protects peripheral and cerebral blood vessels from toxicity of Alzheimer peptide amyloid-beta and inflammatory reactionT Thomas, J A Rhodin, E T Sutton, et al.Shock (Augusta, Ga.)|December 1, 1995
Arteriolar reactivity of endotoxin-tolerant rats after hemorrhage and reinfusionC H Baker, E T Sutton, J M PriceNeuroscience Letters|August 5, 2000
Potassium channel openers prevent beta-amyloid toxicity in bovine vascular endothelial cellsX Chi, E T Sutton, G Hellermann, et al.Neurological Research|July 28, 2001
Physiological levels of beta-amyloid induce cerebral vessel dysfunction and reduce endothelial nitric oxide productionJ M Price, X Chi, G Hellermann, et al.The American Journal of Physiology|October 1, 1993
Effect of muscle length on the in vitro comparison of femoral arteries before and after endotoxin shockZ Zhou, J M Price, E T Sutton, et al.Pageof 130