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J Mosbacher

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Antisense & Nucleic Acid Drug Development|July 12, 2001
Specific inhibition of the rat ligand-gated ion channel P2X3 function via methoxyethoxy-modified phosphorothioated antisense oligonucleotidesG Dorn, S Abdel'Al, F J Natt, et al.
The Journal of Biological Chemistry|January 13, 2001
Control of electrical activity in central neurons by modulating the gating of small conductance Ca2+-activated K+ channelsP Pedarzani, J Mosbacher, A Rivard, et al.
Annals of the New York Academy of Sciences|July 22, 1999
Processing of GABABR1 in heterologous expression systemsJ Mosbacher, K Kaupmann, V Schuler, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 9, 1998
Human gamma-aminobutyric acid type B receptors are differentially expressed and regulate inwardly rectifying K+ channelsK Kaupmann, V Schuler, J Mosbacher, et al.
Science (New York, N.Y.)|December 9, 1994
Control of kinetic properties of AMPA receptor channels by nuclear RNA editingH Lomeli, J Mosbacher, T Melcher, et al.
Nature|January 1, 1999
GABA(B)-receptor subtypes assemble into functional heteromeric complexesK Kaupmann, B Malitschek, V Schuler, et al.
Molecular Pharmacology|February 29, 2000
Ca(2+) requirement for high-affinity gamma-aminobutyric acid (GABA) binding at GABA(B) receptors: involvement of serine 269 of the GABA(B)R1 subunitT Galvez, S Urwyler, L Prézeau, et al.
Molecular Pharmacology|July 27, 1999
The N-terminal domain of gamma-aminobutyric Acid(B) receptors is sufficient to specify agonist and antagonist bindingB Malitschek, C Schweizer, M Keir, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|February 13, 2001
C-terminal interaction is essential for surface trafficking but not for heteromeric assembly of GABA(b) receptorsA Pagano, G Rovelli, J Mosbacher, et al.
Neuron|August 11, 2001
Epilepsy, hyperalgesia, impaired memory, and loss of pre- and postsynaptic GABA(B) responses in mice lacking GABA(B(1))V Schuler, C Lüscher, C Blanchet, et al.
Pageof 2

Showing results (11-20 of 20) with videos related to

Sort By:
Pageof 2
You have reached the last page of results.This site can display upto 20 results.
Antisense & Nucleic Acid Drug Development|July 12, 2001
Specific inhibition of the rat ligand-gated ion channel P2X3 function via methoxyethoxy-modified phosphorothioated antisense oligonucleotidesG Dorn, S Abdel'Al, F J Natt, et al.
The Journal of Biological Chemistry|January 13, 2001
Control of electrical activity in central neurons by modulating the gating of small conductance Ca2+-activated K+ channelsP Pedarzani, J Mosbacher, A Rivard, et al.
Annals of the New York Academy of Sciences|July 22, 1999
Processing of GABABR1 in heterologous expression systemsJ Mosbacher, K Kaupmann, V Schuler, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 9, 1998
Human gamma-aminobutyric acid type B receptors are differentially expressed and regulate inwardly rectifying K+ channelsK Kaupmann, V Schuler, J Mosbacher, et al.
Science (New York, N.Y.)|December 9, 1994
Control of kinetic properties of AMPA receptor channels by nuclear RNA editingH Lomeli, J Mosbacher, T Melcher, et al.
Nature|January 1, 1999
GABA(B)-receptor subtypes assemble into functional heteromeric complexesK Kaupmann, B Malitschek, V Schuler, et al.
Molecular Pharmacology|February 29, 2000
Ca(2+) requirement for high-affinity gamma-aminobutyric acid (GABA) binding at GABA(B) receptors: involvement of serine 269 of the GABA(B)R1 subunitT Galvez, S Urwyler, L Prézeau, et al.
Molecular Pharmacology|July 27, 1999
The N-terminal domain of gamma-aminobutyric Acid(B) receptors is sufficient to specify agonist and antagonist bindingB Malitschek, C Schweizer, M Keir, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|February 13, 2001
C-terminal interaction is essential for surface trafficking but not for heteromeric assembly of GABA(b) receptorsA Pagano, G Rovelli, J Mosbacher, et al.
Neuron|August 11, 2001
Epilepsy, hyperalgesia, impaired memory, and loss of pre- and postsynaptic GABA(B) responses in mice lacking GABA(B(1))V Schuler, C Lüscher, C Blanchet, et al.
Pageof 2