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The American Review of Respiratory Disease|August 1, 1993
Tumor necrosis factor and endotoxin do not directly affect in vitro diaphragm functionP T Diaz, M W Julian, M D Wewers, et al.
The American Review of Respiratory Disease|February 1, 1987
Evaluation of tamoxifen as a therapy to augment alpha-1-antitrypsin concentrations in Z homozygous alpha-1-antitrypsin-deficient subjectsM D Wewers, M L Brantly, M A Casolaro, et al.
The American Journal of Physiology|August 1, 1993
Endotoxin priming of monocytes augments Fc gamma receptor cross-linking-induced TNF-alpha and IL-1 beta releaseG C Kindt, S A Moore, Z W She, et al.
The Journal of Clinical Investigation|September 18, 1997
Antineutrophil cytoplasmic antibodies induce monocyte IL-8 release. Role of surface proteinase-3, alpha1-antitrypsin, and Fcgamma receptorsD R Ralston, C B Marsh, M P Lowe, et al.
American Journal of Respiratory Cell and Molecular Biology|May 5, 1999
Extracellular regulation of interleukin (IL)-1beta through lung epithelial cells and defective IL-1 type II receptor expressionK R Coulter, M D Wewers, M P Lowe, et al.
American Journal of Respiratory Cell and Molecular Biology|October 1, 1993
Tumor necrosis factor increases the elastolytic potential of adherent neutrophils: a role for hypochlorous acidZ W She, M D Wewers, D J Herzyk, et al.
Journal of Immunology (Baltimore, Md. : 1950)|September 15, 1996
Monocyte IL-8 release is induced by two independent Fc gamma R- mediated pathwaysC B Marsh, C L Anderson, M P Lowe, et al.
The American Journal of Physiology|July 1, 1994
IL-1ra suppresses endotoxin-induced IL-1 beta and TNF-alpha release from mononuclear phagocytesC B Marsh, S A Moore, H A Pope, et al.
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