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J N Whitaker

Showing results (191-200 of 215) with videos related to

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Journal of Neuroimmunology|July 1, 1989
Opsonization of normal myelin by anti-myelin antibodies and normal serumP Z Goldenberg, E E Kwon, J A Benjamins, et al.
Journal of Neuroimmunology|July 1, 1993
The structure of a myelin basic protein-associated idiotopeC C Maier, R D LeBoeuf, S R Zhou, et al.
Journal of Clinical Oncology : Official Journal of the American Society of Clinical Oncology|December 1, 1983
Serial cranial computed-tomography scans in children with leukemia given two different forms of central nervous system therapyJ J Ochs, L S Parvey, J N Whitaker, et al.
Neurology|January 1, 1996
Management of patients receiving interferon beta-1b for multiple sclerosis: report of a consensus conferenceF D Lublin, J N Whitaker, B H Eidelman, et al.
Annals of the New York Academy of Sciences|January 1, 1981
Recently recognized congenital myasthenic syndromes: (a) end-plate acetylcholine (ACh) esterase deficiency (b) putative abnormality of the ACh induced ion channel (c) putative defect of ACh resynthesis or mobilization - clinical features, ultrastructure and cytochemistryA G Engel, E H Lambert, D M Mulder, et al.
Neurology|May 10, 2000
Linomide in relapsing and secondary progressive MS: part I: trial design and clinical results. North American Linomide InvestigatorsJ H Noseworthy, J S Wolinsky, F D Lublin, et al.
Archives of Neurology|June 16, 1999
Urinary myelin basic protein-like material in patients with multiple sclerosis during interferon beta-1b treatmentJ N Whitaker, B A Layton, A A Bartolucci, et al.
Transactions of the American Neurological Association|January 1, 1978
Immunoreactive myelin basic protein in the cerebrospinal fluid in neurological disordersJ N Whitaker, R P Lisak, R M Bashir, et al.
Annals of Neurology|June 1, 1982
A newly recognized congenital myasthenic syndrome attributed to a prolonged open time of the acetylcholine-induced ion channelA G Engel, E H Lambert, D M Mulder, et al.
Transactions of the American Neurological Association|January 1, 1979
Investigations of 3 cases of a newly recognized familial, congenital myasthenic syndromeA G Engel, E H Lambert, D M Mulder, et al.
Pageof 22

Showing results (191-200 of 215) with videos related to

Sort By:
Pageof 22
Journal of Neuroimmunology|July 1, 1989
Opsonization of normal myelin by anti-myelin antibodies and normal serumP Z Goldenberg, E E Kwon, J A Benjamins, et al.
Journal of Neuroimmunology|July 1, 1993
The structure of a myelin basic protein-associated idiotopeC C Maier, R D LeBoeuf, S R Zhou, et al.
Journal of Clinical Oncology : Official Journal of the American Society of Clinical Oncology|December 1, 1983
Serial cranial computed-tomography scans in children with leukemia given two different forms of central nervous system therapyJ J Ochs, L S Parvey, J N Whitaker, et al.
Neurology|January 1, 1996
Management of patients receiving interferon beta-1b for multiple sclerosis: report of a consensus conferenceF D Lublin, J N Whitaker, B H Eidelman, et al.
Annals of the New York Academy of Sciences|January 1, 1981
Recently recognized congenital myasthenic syndromes: (a) end-plate acetylcholine (ACh) esterase deficiency (b) putative abnormality of the ACh induced ion channel (c) putative defect of ACh resynthesis or mobilization - clinical features, ultrastructure and cytochemistryA G Engel, E H Lambert, D M Mulder, et al.
Neurology|May 10, 2000
Linomide in relapsing and secondary progressive MS: part I: trial design and clinical results. North American Linomide InvestigatorsJ H Noseworthy, J S Wolinsky, F D Lublin, et al.
Archives of Neurology|June 16, 1999
Urinary myelin basic protein-like material in patients with multiple sclerosis during interferon beta-1b treatmentJ N Whitaker, B A Layton, A A Bartolucci, et al.
Transactions of the American Neurological Association|January 1, 1978
Immunoreactive myelin basic protein in the cerebrospinal fluid in neurological disordersJ N Whitaker, R P Lisak, R M Bashir, et al.
Annals of Neurology|June 1, 1982
A newly recognized congenital myasthenic syndrome attributed to a prolonged open time of the acetylcholine-induced ion channelA G Engel, E H Lambert, D M Mulder, et al.
Transactions of the American Neurological Association|January 1, 1979
Investigations of 3 cases of a newly recognized familial, congenital myasthenic syndromeA G Engel, E H Lambert, D M Mulder, et al.
Pageof 22